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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Molecular insights into the premature aging disease progeria
Sandra Vidak, Roland Foisner
Histochemistry and Cell Biology (2016) Vol. 145, Iss. 4, pp. 401-417
Open Access | Times Cited: 100

Showing 26-50 of 100 citing articles:

Connective Tissue and Age-Related Diseases
Carolyn Ann Sarbacher, Jaroslava Halper
Sub-cellular biochemistry/Subcellular biochemistry (2019), pp. 281-310
Closed Access | Times Cited: 37
SAMMY-seq reveals early alteration of heterochromatin and deregulation of bivalent genes in Hutchinson-Gilford Progeria Syndrome
Endre Sebestyén, Fabrizia Marullo, Federica Lucini, et al.
Nature Communications (2020) Vol. 11, Iss. 1
Open Access | Times Cited: 34
Progerin phosphorylation in interphase is lower and less mechanosensitive than lamin-A,C in iPS-derived mesenchymal stem cells
Sang-Kyun Cho, Amal Abbas, Jerome Irianto, et al.
Nucleus (2018) Vol. 9, Iss. 1, pp. 235-250
Open Access | Times Cited: 36
Protein sequestration at the nuclear periphery as a potential regulatory mechanism in premature aging
Leonid Serebryannyy, Tom Misteli
The Journal of Cell Biology (2017) Vol. 217, Iss. 1, pp. 21-37
Open Access | Times Cited: 34
Cannabinoids as anti-ROS in aged pancreatic islet cells
Maryam Baeeri, Mahban Rahimifard, Seyed Mojtaba Daghighi, et al.
Life Sciences (2020) Vol. 256, pp. 117969-117969
Open Access | Times Cited: 29
Hutchinson-Gilford Progeria Syndrome: An Overview of the Molecular Mechanism, Pathophysiology and Therapeutic Approach
Md. Mominur Rahman, Kazi Sayma Ferdous, Muniruddin Ahmed, et al.
Current Gene Therapy (2021) Vol. 21, Iss. 3, pp. 216-229
Closed Access | Times Cited: 24
Aged induced pluripotent stem cell (iPSCs) as a new cellular model for studying premature aging
Stefania Petrini, Rossella Borghi, Valentina Doria, et al.
Aging (2017) Vol. 9, Iss. 5, pp. 1453-1469
Open Access | Times Cited: 32
Vascular smooth muscle cell loss underpins the accelerated atherosclerosis in Hutchinson-Gilford progeria syndrome
Magda R. Hamczyk, Vicente Andrés
Nucleus (2019) Vol. 10, Iss. 1, pp. 48-54
Open Access | Times Cited: 27
Vascular Smooth Muscle Cell-Specific Progerin Expression Provokes Contractile Impairment in a Mouse Model of Hutchinson-Gilford Progeria Syndrome that Is Ameliorated by Nitrite Treatment
Lara del Campo, Amanda Sánchez‐López, Cristina González‐Gómez, et al.
Cells (2020) Vol. 9, Iss. 3, pp. 656-656
Open Access | Times Cited: 27
Lamin A/C and PI(4,5)P2—A Novel Complex in the Cell Nucleus
Sara Escudeiro-Lopes, Vlada Filimonenko, Lenka Jarolimová, et al.
Cells (2024) Vol. 13, Iss. 5, pp. 399-399
Open Access | Times Cited: 3
Upregulation of the aging related LMNA splice variant progerin in dilated cardiomyopathy
Moritz Messner, Santhosh Kumar Ghadge, Valentina Goetsch, et al.
PLoS ONE (2018) Vol. 13, Iss. 4, pp. e0196739-e0196739
Open Access | Times Cited: 27
Blank spots on the map: some current questions on nuclear organization and genome architecture
Carmen Adriaens, Leonid Serebryannyy, Marina Feric, et al.
Histochemistry and Cell Biology (2018) Vol. 150, Iss. 6, pp. 579-592
Open Access | Times Cited: 27
Emerging candidate treatment strategies for Hutchinson-Gilford progeria syndrome
Charlotte Strandgren, Gwladys Revêchon, Agustín Sola-Carvajal, et al.
Biochemical Society Transactions (2017) Vol. 45, Iss. 6, pp. 1279-1293
Closed Access | Times Cited: 25
Evaluation of musculoskeletal phenotype of the G608G progeria mouse model with lonafarnib, pravastatin, and zoledronic acid as treatment groups
M. Belén Cubria, Sebastián Suárez, Aidin Masoudi, et al.
Proceedings of the National Academy of Sciences (2020) Vol. 117, Iss. 22, pp. 12029-12040
Open Access | Times Cited: 23
Autophagic Removal of Farnesylated Carboxy-Terminal Lamin Peptides
Xiang Lu, Karima Djabali
Cells (2018) Vol. 7, Iss. 4, pp. 33-33
Open Access | Times Cited: 23
Lamins and bone disorders: current understanding and perspectives
Chiara Gargiuli, Elisa Schena, Elisabetta Mattioli, et al.
Oncotarget (2018) Vol. 9, Iss. 32, pp. 22817-22831
Open Access | Times Cited: 21
Barrier-to-autointegration factor: a first responder for repair of nuclear ruptures
Charles Halfmann, Kyle J. Roux
Cell Cycle (2021) Vol. 20, Iss. 7, pp. 647-660
Open Access | Times Cited: 17
OGT (O-GlcNAc Transferase) Selectively Modifies Multiple Residues Unique to Lamin A
Dan N. Simon, Amanda Wriston, Qiong Fan, et al.
Cells (2018) Vol. 7, Iss. 5, pp. 44-44
Open Access | Times Cited: 20
In focus in HCB
Douglas J. Taatjes, Jürgen Roth
Histochemistry and Cell Biology (2017) Vol. 147, Iss. 3, pp. 303-305
Open Access | Times Cited: 19
Next-Generation Sequencing and Quantitative Proteomics of Hutchinson-Gilford progeria syndrome-derived cells point to a role of nucleotide metabolism in premature aging
Jesús Mateos, Juan Fafián‐Labora, Miriam Morente-López, et al.
PLoS ONE (2018) Vol. 13, Iss. 10, pp. e0205878-e0205878
Open Access | Times Cited: 19
Pharmacotherapy to gene editing: potential therapeutic approaches for Hutchinson–Gilford progeria syndrome
Saurabh Saxena, Sanjeev Kumar
GeroScience (2020) Vol. 42, Iss. 2, pp. 467-494
Open Access | Times Cited: 18
Towards delineating the chain of events that cause premature senescence in the accelerated aging syndrome Hutchinson–Gilford progeria (HGPS)
Oliver Dreesen
Biochemical Society Transactions (2020) Vol. 48, Iss. 3, pp. 981-991
Open Access | Times Cited: 17
Nanoparticle-Based Combination Therapy for Melanoma
Hongbo Chen, Kai Hou, Jing Yu, et al.
Frontiers in Oncology (2022) Vol. 12
Open Access | Times Cited: 10
Nucleoplasmic lamins define growth-regulating functions of lamina-associated polypeptide 2α in progeria cells
Sandra Vidak, Konstantina Georgiou, Petra Fichtinger, et al.
Journal of Cell Science (2017)
Open Access | Times Cited: 18
Hutchinson-Gilford Progeria Syndrome: Challenges at Bench and Bedside
Ray Kreienkamp, Susana Gonzalo
Sub-cellular biochemistry/Subcellular biochemistry (2019), pp. 435-451
Closed Access | Times Cited: 17

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