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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

PINK1 kinase dysfunction triggers neurodegeneration in the primate brain without impacting mitochondrial homeostasis
Weili Yang, Xiangyu Guo, Zhuchi Tu, et al.
Protein & Cell (2021) Vol. 13, Iss. 1, pp. 26-46
Open Access | Times Cited: 49

Showing 26-50 of 49 citing articles:

Mitochondrial-Dependent and Independent Functions of PINK1
Xiusheng Chen, Qi Wang, Shihua Li, et al.
Frontiers in Cell and Developmental Biology (2022) Vol. 10
Open Access | Times Cited: 14
Differential distribution of PINK1 and Parkin in the primate brain implies distinct roles
Yanting Liu, Wei Huang, Jiayi Wen, et al.
Neural Regeneration Research (2024) Vol. 20, Iss. 4, pp. 1124-1134
Open Access | Times Cited: 2
Development of CRISPR Cas9, spin-off technologies and their application in model construction and potential therapeutic methods of Parkinson’s disease
Jiangbo Qu, Na Liu, Lu Gao, et al.
Frontiers in Neuroscience (2023) Vol. 17
Open Access | Times Cited: 6
Modeling neurodegenerative diseases using non-human primates: advances and challenges
Bang Li, Dajian He, Xiao‐Jiang Li, et al.
Ageing and Neurodegenerative Diseases (2022) Vol. 2, Iss. 3, pp. 12-12
Open Access | Times Cited: 9
Comparative analysis of primate and pig cells reveals primate-specific PINK1 expression and phosphorylation
Xiusheng Chen, Rui Han, Yanting Liu, et al.
动物学研究 (2023) Vol. 45, Iss. 2, pp. 242-252
Open Access | Times Cited: 5
Differential expression and roles of Huntingtin and Huntingtin-associated protein 1 in the mouse and primate brains
Xing‐Xing Chen, Yize Sun, Laiqiang Chen, et al.
Cellular and Molecular Life Sciences (2022) Vol. 79, Iss. 11
Closed Access | Times Cited: 8
New perspectives on the role of mitochondria in Parkinson's disease
Shiyi Yin, Yongjiang Zhang, Jiannan Wu, et al.
Journal of Neurorestoratology (2024) Vol. 12, Iss. 2, pp. 100112-100112
Open Access | Times Cited: 1
PINK1 mediates neuronal survival in monkey
Z. Sun, Jiangyu Ye, Junying Yuan
Protein & Cell (2021) Vol. 13, Iss. 1, pp. 4-5
Open Access | Times Cited: 10
Progress in Parkinson's disease animal models of genetic defects: Characteristics and application
Chutian Zhang, Shiya Chen, Xiyu Li, et al.
Biomedicine & Pharmacotherapy (2022) Vol. 155, pp. 113768-113768
Open Access | Times Cited: 6
Brain Region- and Age-Dependent 5-Hydroxymethylcytosine Activity in the Non-Human Primate
Yanru Xu, Liying Zhong, Huixian Wei, et al.
Frontiers in Aging Neuroscience (2022) Vol. 14
Open Access | Times Cited: 5
Genetically engineered pig models of neurological diseases
Caijuan Li, Jun Li, Liangxue Lai, et al.
Ageing and Neurodegenerative Diseases (2022) Vol. 2, Iss. 3, pp. 13-13
Open Access | Times Cited: 5
PINK1 deficiency with Ca2+ changes in the hippocampus exacerbates septic encephalopathy in mice
Cui Li, Tianyu Yu, Wenxing Li, et al.
Chemico-Biological Interactions (2023) Vol. 374, pp. 110413-110413
Closed Access | Times Cited: 2
CRISPR/Cas9: implication for modeling and therapy of amyotrophic lateral sclerosis
Yajun Shi, Yan Zhao, Likui Lu, et al.
Frontiers in Neuroscience (2023) Vol. 17
Open Access | Times Cited: 2
The causal effects of intelligence and fluid intelligence on Parkinson’s disease: a Mendelian randomization study
聡 柏井, Xiaojiao Zhong, XuLi Min, et al.
Frontiers in Aging Neuroscience (2024) Vol. 16
Open Access
Naturally occurring hyperactive variants of human parkin
Tahrima Saiha Huq, Jean Luo, Rayan Fakih, et al.
Communications Biology (2024) Vol. 7, Iss. 1
Open Access
Ideal animal models according to multifaceted mechanisms and peculiarities in neurological disorders: present and challenges
Sandesh Lamichhane, Jo‐Eun Seo, Ji Hoon Jeong, et al.
Archives of Pharmacal Research (2024)
Closed Access
Animal models for research on neurodegenerative diseases
Xiao‐Jiang Li
Ageing and Neurodegenerative Diseases (2023) Vol. 3, Iss. 3
Open Access | Times Cited: 1
Disruption of Dopamine Homeostasis Associated with Alteration of Proteins in Synaptic Vesicles: A Putative Central Mechanism of Parkinson’s Disease Pathogenesis
Xuanxiang Jin, Xiaoli Si, Xiaoguang Lei, et al.
Aging and Disease (2023)
Open Access | Times Cited: 1
Neuronal activity in the anterior paraventricular nucleus of thalamus positively correlated with sweetener consumption in mice
Shaolei Jiang, Bo Song, Zhongdong Liu, et al.
Neuroscience Research (2024) Vol. 205, pp. 16-26
Open Access
Characterization of a new set of monoclonal β-amyloid antibodies
Huan-Yu Che, Jia-Qi Ai, Yang Chen, et al.
Human Brain (2022) Vol. 1, Iss. 1
Open Access | Times Cited: 2
CRISPR: a tool with potential for genomic reprogramming in neurological disorders
Yogesh K. Dhuriya, Aijaz Ahmad Naik
Molecular Biology Reports (2022) Vol. 50, Iss. 2, pp. 1845-1856
Closed Access | Times Cited: 2
Expanding Views of Mitochondria in Parkinson’s Disease: Focusing on PINK1 and GBA1 Mutations
Yu Yuan, Xizhen Ma, Ning Song, et al.
Neuroscience Bulletin (2022) Vol. 38, Iss. 7, pp. 825-828
Open Access | Times Cited: 1
Recent advances in genetically modified large-animal models of human diseases
Jing Zhang, Xiaoyue Sun, Chunwei Cao
BIO Integration (2022) Vol. 3, Iss. 4
Open Access | Times Cited: 1
PINK1 and BNIP3 mitophagy inducers have an antagonistic effect on Rbf1-induced apoptosis in Drosophila.
Mélanie Fages, Vincent Ruby, Mégane Brusson, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2023)
Open Access
Neuropathological insights from SHANK3 mutant animal models
Jiawei Zhang, Dajian He, Xiao‐Jiang Li
Ageing and Neurodegenerative Diseases (2023) Vol. 3, Iss. 4
Open Access
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