OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!
If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.
Requested Article:
miR-199a-3p promotes cardiomyocyte proliferation by inhibiting Cd151 expression
Tao Ying, Hongjie Zhang, Shiqi Huang, et al.
Biochemical and Biophysical Research Communications (2019) Vol. 516, Iss. 1, pp. 28-36
Closed Access | Times Cited: 35
Tao Ying, Hongjie Zhang, Shiqi Huang, et al.
Biochemical and Biophysical Research Communications (2019) Vol. 516, Iss. 1, pp. 28-36
Closed Access | Times Cited: 35
Showing 26-50 of 35 citing articles:
MicroRNA-219c-5p Regulates Bladder Fibrosis by Targeting FN1
Bowen Liu, Yafei Ding, Peng Li, et al.
Research Square (Research Square) (2020)
Open Access | Times Cited: 7
Bowen Liu, Yafei Ding, Peng Li, et al.
Research Square (Research Square) (2020)
Open Access | Times Cited: 7
Adaptation of cardiomyogenesis to the generation and maturation of cardiomyocytes from human pluripotent stem cells
Alicja Martyniak, Mateusz Jeż, Józef Dulak, et al.
IUBMB Life (2022) Vol. 75, Iss. 1, pp. 8-29
Closed Access | Times Cited: 4
Alicja Martyniak, Mateusz Jeż, Józef Dulak, et al.
IUBMB Life (2022) Vol. 75, Iss. 1, pp. 8-29
Closed Access | Times Cited: 4
Targeting cardiomyocyte proliferation as a key approach of promoting heart repair after injury
Shuainan Li, Wenya Ma, Benzhi Cai
Molecular Biomedicine (2021) Vol. 2, Iss. 1
Open Access | Times Cited: 5
Shuainan Li, Wenya Ma, Benzhi Cai
Molecular Biomedicine (2021) Vol. 2, Iss. 1
Open Access | Times Cited: 5
Non-coding RNAs to regulate cardiomyocyte proliferation: A new trend in therapeutic cardiac regeneration
Kele Qin, Xiaohui Xie, Weijie Tang, et al.
Frontiers in Cardiovascular Medicine (2022) Vol. 9
Open Access | Times Cited: 3
Kele Qin, Xiaohui Xie, Weijie Tang, et al.
Frontiers in Cardiovascular Medicine (2022) Vol. 9
Open Access | Times Cited: 3
CircSorbs1 regulates myocardial regeneration and reduces cancer therapy-related cardiovascular toxicity through the Mir-99/GATA4 pathway
Kang Huang, Denggao Huang, Qiang Li, et al.
Discover Oncology (2024) Vol. 15, Iss. 1
Open Access
Kang Huang, Denggao Huang, Qiang Li, et al.
Discover Oncology (2024) Vol. 15, Iss. 1
Open Access
<italic>miR-199a-3p</italic> suppresses <italic>Vldlr</italic> expression to promote cardiomyocyte proliferation
Rui Jiang, Lijuan Pei, Hongjie Zhang, et al.
Acta Biochimica et Biophysica Sinica (2024)
Open Access
Rui Jiang, Lijuan Pei, Hongjie Zhang, et al.
Acta Biochimica et Biophysica Sinica (2024)
Open Access
MicroRNA Expression in the Infarcted Heart Following Neonatal Cardiovascular Progenitor Cell Transplantation in a Sheep Model of Stem Cell–Based Repair
Larry V. Lopez, Victor Camberos, Leonard L. Bailey, et al.
Cell Transplantation (2022) Vol. 31
Open Access | Times Cited: 1
Larry V. Lopez, Victor Camberos, Leonard L. Bailey, et al.
Cell Transplantation (2022) Vol. 31
Open Access | Times Cited: 1
KDM5B as a potential epigenetic regulator of cardiomyocyte regeneration and cell cycle activation.
Dana Hammouri
(2023)
Open Access
Dana Hammouri
(2023)
Open Access
MicroRNA-219c-5p regulates bladder smooth muscle cell fibrosis by targeting FN1
Bowen Liu, Yafei Ding, Peng Li, et al.
Research Square (Research Square) (2020)
Open Access
Bowen Liu, Yafei Ding, Peng Li, et al.
Research Square (Research Square) (2020)
Open Access
Influencing factors and evaluation methods of cardiomyocyte proliferation
Wenbin Fu, Wei Wang, Chunyu Zeng
Scientia Sinica Vitae (2021) Vol. 52, Iss. 2, pp. 161-173
Open Access
Wenbin Fu, Wei Wang, Chunyu Zeng
Scientia Sinica Vitae (2021) Vol. 52, Iss. 2, pp. 161-173
Open Access
