Open Access Helper

OpenAlex Citation Counts

OpenAlex Citations Logo

OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Sequestration of multiple RNA recognition motif-containing proteins by C9orf72 repeat expansions
Johnathan Cooper‐Knock, Matthew J. Walsh, Adrian Higginbottom, et al.
Brain (2014) Vol. 137, Iss. 7, pp. 2040-2051
Open Access | Times Cited: 288

Showing 26-50 of 288 citing articles:

p53 is a central regulator driving neurodegeneration caused by C9orf72 poly(PR)
Maya Maor-Nof, Zohar Shipony, Rodrigo López‐González, et al.
Cell (2021) Vol. 184, Iss. 3, pp. 689-708.e20
Open Access | Times Cited: 141
C9ORF72: What It Is, What It Does, and Why It Matters
Julie Smeyers, Elena-Gaia Banchi, Morwena Latouche
Frontiers in Cellular Neuroscience (2021) Vol. 15
Open Access | Times Cited: 113
Genome-wide identification of the genetic basis of amyotrophic lateral sclerosis
Sai Zhang, Johnathan Cooper‐Knock, Annika K. Weimer, et al.
Neuron (2022) Vol. 110, Iss. 6, pp. 992-1008.e11
Open Access | Times Cited: 98
Neuromuscular organoids model spinal neuromuscular pathologies in C9orf72 amyotrophic lateral sclerosis
Chong Gao, Qinghua Shi, Pan Xue, et al.
Cell Reports (2024) Vol. 43, Iss. 3, pp. 113892-113892
Open Access | Times Cited: 18
RNA dysregulation in neurodegenerative diseases
Yini Li, Shuying Sun
The EMBO Journal (2025) Vol. 44, Iss. 3, pp. 613-638
Open Access | Times Cited: 3
Amyotrophic lateral sclerosis caused by hexanucleotide repeat expansions in C9orf72: from genetics to therapeutics
Sarah Mizielinska, Guillaume M. Hautbergue, Tania F. Gendron, et al.
The Lancet Neurology (2025) Vol. 24, Iss. 3, pp. 261-274
Closed Access | Times Cited: 3
Distribution of dipeptide repeat proteins in cellular models and C9orf72 mutation cases suggests link to transcriptional silencing
Martin H. Schludi, Stephanie May, Friedrich A. Grässer, et al.
Acta Neuropathologica (2015) Vol. 130, Iss. 4, pp. 537-555
Open Access | Times Cited: 175
Familial Amyotrophic Lateral Sclerosis
Khrista Boylan
Neurologic Clinics (2015) Vol. 33, Iss. 4, pp. 807-830
Open Access | Times Cited: 166
Dysregulated molecular pathways in amyotrophic lateral sclerosis–frontotemporal dementia spectrum disorder
Fen‐Biao Gao, Sandra Almeida, Rodrigo López‐González
The EMBO Journal (2017) Vol. 36, Iss. 20, pp. 2931-2950
Open Access | Times Cited: 166
C9orf72 expansion disrupts ATM-mediated chromosomal break repair
Callum Walker, Saúl Herranz-Martín, Evangelia Karyka, et al.
Nature Neuroscience (2017) Vol. 20, Iss. 9, pp. 1225-1235
Open Access | Times Cited: 162
Antisense RNA foci in the motor neurons of C9ORF72-ALS patients are associated with TDP-43 proteinopathy
Johnathan Cooper‐Knock, Adrian Higginbottom, Matthew J. Stopford, et al.
Acta Neuropathologica (2015) Vol. 130, Iss. 1, pp. 63-75
Open Access | Times Cited: 159
A complex of C9ORF72 and p62 uses arginine methylation to eliminate stress granules by autophagy
Maneka Chitiprolu, Chantal Jagow, Véronique Tremblay, et al.
Nature Communications (2018) Vol. 9, Iss. 1
Open Access | Times Cited: 155
There has been an awakening: Emerging mechanisms of C9orf72 mutations in FTD/ALS
Aaron D. Gitler, Hitomi Tsuiji
Brain Research (2016) Vol. 1647, pp. 19-29
Open Access | Times Cited: 148
Genetics of FTLD: overview and what else we can expect from genetic studies
Cyril Pottier, Thomas A. Ravenscroft, Mónica Sánchez-Contreras, et al.
Journal of Neurochemistry (2016) Vol. 138, Iss. S1, pp. 32-53
Open Access | Times Cited: 138
Frontotemporal lobar degeneration: Pathogenesis, pathology and pathways to phenotype
David Mann, Julie S. Snowden
Brain Pathology (2017) Vol. 27, Iss. 6, pp. 723-736
Open Access | Times Cited: 137
SRSF1-dependent nuclear export inhibition of C9ORF72 repeat transcripts prevents neurodegeneration and associated motor deficits
Guillaume M. Hautbergue, Lydia M. Castelli, Laura Ferraiuolo, et al.
Nature Communications (2017) Vol. 8, Iss. 1
Open Access | Times Cited: 130
RNA metabolism in neurodegenerative disease
Elaine Y. Liu, Christopher P. Cali, Edward B. Lee
Disease Models & Mechanisms (2017) Vol. 10, Iss. 5, pp. 509-518
Open Access | Times Cited: 125
Review: Neuropathology of non‐tau frontotemporal lobar degeneration
Manuela Neumann, Ian R. Mackenzie
Neuropathology and Applied Neurobiology (2018) Vol. 45, Iss. 1, pp. 19-40
Closed Access | Times Cited: 123
Inside out: the role of nucleocytoplasmic transport in ALS and FTLD
Steven Boeynaems, Elke Bogaert, Philip Van Damme, et al.
Acta Neuropathologica (2016) Vol. 132, Iss. 2, pp. 159-173
Open Access | Times Cited: 120
A zebrafish model for C9orf72 ALS reveals RNA toxicity as a pathogenic mechanism
Bart Swinnen, André Bento‐Abreu, Tania F. Gendron, et al.
Acta Neuropathologica (2018) Vol. 135, Iss. 3, pp. 427-443
Open Access | Times Cited: 120
Reduced autophagy upon C9ORF72 loss synergizes with dipeptide repeat protein toxicity in G4C2 repeat expansion disorders
Manon Boivin, Véronique Pfister, Angéline Gaucherot, et al.
The EMBO Journal (2020) Vol. 39, Iss. 4
Open Access | Times Cited: 120
Biomarkers in Motor Neuron Disease: A State of the Art Review
Nick Verber, Stephanie Shepheard, Matilde Sassani, et al.
Frontiers in Neurology (2019) Vol. 10
Open Access | Times Cited: 116
Extensive size variability of the GGGGCC expansion in C9orf72 in both neuronal and non-neuronal tissues in 18 patients with ALS or FTD
Angelica Nordin, Chizuru Akimoto, Anna Wuolikainen, et al.
Human Molecular Genetics (2015) Vol. 24, Iss. 11, pp. 3133-3142
Open Access | Times Cited: 110
Axonal transport defects are a common phenotype inDrosophilamodels of ALS
Katie R. Baldwin, Vinay K. Godena, Victoria L. Hewitt, et al.
Human Molecular Genetics (2016), pp. ddw105-ddw105
Open Access | Times Cited: 109
Nuclear accumulation of mRNAs underlies G4C2 repeat-induced translational repression in a cellular model of C9orf72 ALS
Simona Rossi, Alessia Serrano, Valeria Gerbino, et al.
Journal of Cell Science (2015)
Open Access | Times Cited: 108

Scroll to top