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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Humanized mutant FUS drives progressive motor neuron degeneration without aggregation in ‘FUSDelta14’ knockin mice
Anny Devoy, Bernadett Kalmár, Michelle Stewart, et al.
Brain (2017) Vol. 140, Iss. 11, pp. 2797-2805
Open Access | Times Cited: 104

Showing 26-50 of 104 citing articles:

Maximizing the translational potential of neurophysiology in amyotrophic lateral sclerosis: a study on compound muscle action potentials
S. J. McKinnon, Zekai Qiang, Amy Keerie, et al.
Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration (2025), pp. 1-9
Open Access | Times Cited: 1
Mouse models of neurodegeneration: Know your question, know your mouse
Elizabeth Fisher, David M. Bannerman
Science Translational Medicine (2019) Vol. 11, Iss. 493
Open Access | Times Cited: 65
A Systematic Review of Suggested Molecular Strata, Biomarkers and Their Tissue Sources in ALS
Udaya Geetha Vijayakumar, Vanessa Milla, M. Stafford, et al.
Frontiers in Neurology (2019) Vol. 10
Open Access | Times Cited: 65
Aberrant interaction of FUS with the U1 snRNA provides a molecular mechanism of FUS induced amyotrophic lateral sclerosis
Daniel Jutzi, Sébastien Campagne, Ralf Schmidt, et al.
Nature Communications (2020) Vol. 11, Iss. 1
Open Access | Times Cited: 60
Focus on the heterogeneity of amyotrophic lateral sclerosis
Caterina Bendotti, Valentina Bonetto, Elisabetta Pupillo, et al.
Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration (2020) Vol. 21, Iss. 7-8, pp. 485-495
Open Access | Times Cited: 55
FUS-ALS mutants alter FMRP phase separation equilibrium and impair protein translation
Nicol Birsa, Agnieszka M Ule, Maria Giovanna Garone, et al.
Science Advances (2021) Vol. 7, Iss. 30
Open Access | Times Cited: 53
In vivo stress granule misprocessing evidenced in a FUS knock-in ALS mouse model
Xue Zhang, Fengchao Wang, Yi Hu, et al.
Brain (2020) Vol. 143, Iss. 5, pp. 1350-1367
Open Access | Times Cited: 52
Small junction, big problems: Neuromuscular junction pathology in mouse models of amyotrophic lateral sclerosis (ALS)
Abrar Alhindi, Ines Boehm, Helena Chaytow
Journal of Anatomy (2021) Vol. 241, Iss. 5, pp. 1089-1107
Open Access | Times Cited: 51
Cytoplasmic FUS triggers early behavioral alterations linked to cortical neuronal hyperactivity and inhibitory synaptic defects
Jelena Scekic‐Zahirovic, Inmaculada Sanjuan-Ruiz, Vanessa W. Y. Kan, et al.
Nature Communications (2021) Vol. 12, Iss. 1
Open Access | Times Cited: 46
Disrupted autophagy and neuronal dysfunction in C. elegans knockin models of FUS amyotrophic lateral sclerosis
Saba N. Baskoylu, Natalie Chapkis, Burak Unsal, et al.
Cell Reports (2022) Vol. 38, Iss. 4, pp. 110195-110195
Open Access | Times Cited: 31
The prionlike domain of FUS is multiphosphorylated following DNA damage without altering nuclear localization
Shannon N. Rhoads, Zachary Monahan, Debra Yee, et al.
Molecular Biology of the Cell (2018) Vol. 29, Iss. 15, pp. 1786-1797
Open Access | Times Cited: 48
Role of RNA Binding Proteins with prion-like domains in muscle and neuromuscular diseases
Gina Picchiarelli, Luc Dupuis
Cell Stress (2020) Vol. 4, Iss. 4, pp. 76-91
Open Access | Times Cited: 48
IN VITRO AND IN VIVO MODELS OF AMYOTROPHIC LATERAL SCLEROSIS: AN UPDATED OVERVIEW
Auderlan M. Gois, Deise Maria Furtado de Mendonça, Marco Aurélio M. Freire, et al.
Brain Research Bulletin (2020) Vol. 159, pp. 32-43
Open Access | Times Cited: 44
The RNA-binding protein FUS is chaperoned and imported into the nucleus by a network of import receptors
Imke Baade, Saskia Hutten, Erin L. Sternburg, et al.
Journal of Biological Chemistry (2021) Vol. 296, pp. 100659-100659
Open Access | Times Cited: 40
Advances in mouse genetics for the study of human disease
Steve D. M. Brown
Human Molecular Genetics (2021) Vol. 30, Iss. R2, pp. R274-R284
Open Access | Times Cited: 39
Modelling amyotrophic lateral sclerosis in rodents
Tiffany W. Todd, Leonard Petrucelli
Nature reviews. Neuroscience (2022) Vol. 23, Iss. 4, pp. 231-251
Closed Access | Times Cited: 25
Increased FUS levels in astrocytes leads to astrocyte and microglia activation and neuronal death
Maria Antonietta Ajmone‐Cat, Angela Onori, Camilla Toselli, et al.
Scientific Reports (2019) Vol. 9, Iss. 1
Open Access | Times Cited: 40
Antiviral Immune Response as a Trigger of FUS Proteinopathy in Amyotrophic Lateral Sclerosis
Tatyana A. Shelkovnikova, Haiyan An, Lucy Skelt, et al.
Cell Reports (2019) Vol. 29, Iss. 13, pp. 4496-4508.e4
Open Access | Times Cited: 38
ALS-related FUS mutations alter axon growth in motoneurons and affect HuD/ELAVL4 and FMRP activity
Maria Giovanna Garone, Nicol Birsa, Maria Rosito, et al.
Communications Biology (2021) Vol. 4, Iss. 1
Open Access | Times Cited: 31
FUS(1-359) transgenic mice as a model of ALS: pathophysiological and molecular aspects of the proteinopathy
S. Yu. Funikov, Alexander P. Rezvykh, Pavel Mazin, et al.
Neurogenetics (2018) Vol. 19, Iss. 3, pp. 189-204
Closed Access | Times Cited: 38
Uses for humanised mouse models in precision medicine for neurodegenerative disease
R. Nair, Silvia Corrochano, Samanta Gasco, et al.
Mammalian Genome (2019) Vol. 30, Iss. 7-8, pp. 173-191
Open Access | Times Cited: 35
Neuromuscular disease modeling on a chip
Jeffrey W. Santoso, Megan L. McCain
Disease Models & Mechanisms (2020) Vol. 13, Iss. 7
Open Access | Times Cited: 28
Dysfunction in nonsense-mediated decay, protein homeostasis, mitochondrial function, and brain connectivity in ALS-FUS mice with cognitive deficits
Wan Yun Ho, Ira Agrawal, Sheue-Houy Tyan, et al.
Acta Neuropathologica Communications (2021) Vol. 9, Iss. 1
Open Access | Times Cited: 25
ALS-linked cytoplasmic FUS assemblies are compositionally different from physiological stress granules and sequester hnRNPA3, a novel modifier of FUS toxicity
Haiyan An, Gioana Litscher, Naruaki Watanabe, et al.
Neurobiology of Disease (2021) Vol. 162, pp. 105585-105585
Open Access | Times Cited: 25
Artificial intelligence for neurodegenerative experimental models
Sarah J. Marzi, Brian M. Schilder, Alexi Nott, et al.
Alzheimer s & Dementia (2023) Vol. 19, Iss. 12, pp. 5970-5987
Open Access | Times Cited: 11

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