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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Expanded GGGGCC repeat RNA associated with amyotrophic lateral sclerosis and frontotemporal dementia causes neurodegeneration
Zihui Xu, Mickaël Poidevin, Xuekun Li, et al.
Proceedings of the National Academy of Sciences (2013) Vol. 110, Iss. 19, pp. 7778-7783
Open Access | Times Cited: 335

Showing 51-75 of 335 citing articles:

Dysregulated molecular pathways in amyotrophic lateral sclerosis–frontotemporal dementia spectrum disorder
Fen‐Biao Gao, Sandra Almeida, Rodrigo López‐González
The EMBO Journal (2017) Vol. 36, Iss. 20, pp. 2931-2950
Open Access | Times Cited: 166
Dissection of genetic factors associated with amyotrophic lateral sclerosis
Claire S. Leblond, Hannah Kaneb, Patrick A. Dion, et al.
Experimental Neurology (2014) Vol. 262, pp. 91-101
Closed Access | Times Cited: 160
Microglia and C9orf72 in neuroinflammation and ALS and frontotemporal dementia
Deepti Lall, Robert H. Baloh
Journal of Clinical Investigation (2017) Vol. 127, Iss. 9, pp. 3250-3258
Open Access | Times Cited: 155
There has been an awakening: Emerging mechanisms of C9orf72 mutations in FTD/ALS
Aaron D. Gitler, Hitomi Tsuiji
Brain Research (2016) Vol. 1647, pp. 19-29
Open Access | Times Cited: 148
Mechanisms of toxicity in C9FTLD/ALS
Tania F. Gendron, Véronique Belzil, Yong‐Jie Zhang, et al.
Acta Neuropathologica (2014) Vol. 127, Iss. 3, pp. 359-376
Open Access | Times Cited: 141
Characterization of the dipeptide repeat protein in the molecular pathogenesis of c9FTD/ALS
Mai Yamakawa, Daisuke Ito, Takao Honda, et al.
Human Molecular Genetics (2014) Vol. 24, Iss. 6, pp. 1630-1645
Open Access | Times Cited: 137
From animal models to human disease: a genetic approach for personalized medicine in ALS
Vincent Picher‐Martel, Paul N. Valdmanis, Peter V. Gould, et al.
Acta Neuropathologica Communications (2016) Vol. 4, Iss. 1
Open Access | Times Cited: 135
Pathological mechanisms underlying TDP-43 driven neurodegeneration in FTLD-ALS spectrum disorders
Jonathan Janssens, Christine Van Broeckhoven
Human Molecular Genetics (2013) Vol. 22, Iss. R1, pp. R77-R87
Open Access | Times Cited: 133
RNA Structures as Mediators of Neurological Diseases and as Drug Targets
Viachaslau Bernat, Matthew D. Disney
Neuron (2015) Vol. 87, Iss. 1, pp. 28-46
Open Access | Times Cited: 130
Hypermethylation of repeat expanded C9orf72 is a clinical and molecular disease modifier
Jenny Russ, Elaine Y. Liu, Kathryn Wu, et al.
Acta Neuropathologica (2014) Vol. 129, Iss. 1, pp. 39-52
Open Access | Times Cited: 121
A zebrafish model for C9orf72 ALS reveals RNA toxicity as a pathogenic mechanism
Bart Swinnen, André Bento‐Abreu, Tania F. Gendron, et al.
Acta Neuropathologica (2018) Vol. 135, Iss. 3, pp. 427-443
Open Access | Times Cited: 120
Molecular and Cellular Mechanisms Affected in ALS
Laura Le Gall, Ekene Anakor, Owen Connolly, et al.
Journal of Personalized Medicine (2020) Vol. 10, Iss. 3, pp. 101-101
Open Access | Times Cited: 112
Brain distribution of dipeptide repeat proteins in frontotemporal lobar degeneration and motor neurone disease associated with expansions in C9ORF72
Yvonne S. Davidson, Holly Barker, Andrew Robinson, et al.
Acta Neuropathologica Communications (2014) Vol. 2, Iss. 1
Open Access | Times Cited: 111
Axonal transport defects are a common phenotype inDrosophilamodels of ALS
Katie R. Baldwin, Vinay K. Godena, Victoria L. Hewitt, et al.
Human Molecular Genetics (2016), pp. ddw105-ddw105
Open Access | Times Cited: 109
Nuclear accumulation of mRNAs underlies G4C2 repeat-induced translational repression in a cellular model of C9orf72 ALS
Simona Rossi, Alessia Serrano, Valeria Gerbino, et al.
Journal of Cell Science (2015)
Open Access | Times Cited: 108
Modeling Neurodegenerative Disorders in Drosophila melanogaster
Harris Bolus, Kassi L. Crocker, Grace Boekhoff‐Falk, et al.
International Journal of Molecular Sciences (2020) Vol. 21, Iss. 9, pp. 3055-3055
Open Access | Times Cited: 104
A fruitful endeavor: Modeling ALS in the fruit fly
Ian Casci, Udai Bhan Pandey
Brain Research (2014) Vol. 1607, pp. 47-74
Open Access | Times Cited: 100
RNA toxicity and foci formation in microsatellite expansion diseases
Nan Zhang, Tetsuo Ashizawa
Current Opinion in Genetics & Development (2017) Vol. 44, pp. 17-29
Open Access | Times Cited: 96
Disease Mechanisms ofC9ORF72Repeat Expansions
Tania F. Gendron, Leonard Petrucelli
Cold Spring Harbor Perspectives in Medicine (2017) Vol. 8, Iss. 4, pp. a024224-a024224
Open Access | Times Cited: 90
Traumatic injury induces stress granule formation and enhances motor dysfunctions in ALS/FTD models
Eric N. Anderson, Lauren Gochenaur, Aditi Singh, et al.
Human Molecular Genetics (2018) Vol. 27, Iss. 8, pp. 1366-1381
Open Access | Times Cited: 89
Repeat-associated non-ATG (RAN) translation
John D. Cleary, Amrutha Pattamatta, Laura P.W. Ranum
Journal of Biological Chemistry (2018) Vol. 293, Iss. 42, pp. 16127-16141
Open Access | Times Cited: 88
Targeting RNA-Mediated Toxicity in C9orf72 ALS and/or FTD by RNAi-Based Gene Therapy
Raygene Martier, Jolanda M.P. Liefhebber, Ana Garcı́a-Osta, et al.
Molecular Therapy — Nucleic Acids (2019) Vol. 16, pp. 26-37
Open Access | Times Cited: 79
Nucleocytoplasmic Proteomic Analysis Uncovers eRF1 and Nonsense-Mediated Decay as Modifiers of ALS/FTD C9orf72 Toxicity
Juan A. Ortega, Elizabeth L. Daley, Sukhleen Kour, et al.
Neuron (2020) Vol. 106, Iss. 1, pp. 90-107.e13
Open Access | Times Cited: 78
New pathologic mechanisms in nucleotide repeat expansion disorders
Caitlin M. Rodriguez, Peter K. Todd
Neurobiology of Disease (2019) Vol. 130, pp. 104515-104515
Open Access | Times Cited: 77
Circular RNA Cwc27 contributes to Alzheimer’s disease pathogenesis by repressing Pur-α activity
Chenghuan Song, Yongfang Zhang, Wan‐Ying Huang, et al.
Cell Death and Differentiation (2021) Vol. 29, Iss. 2, pp. 393-406
Open Access | Times Cited: 71

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