Open Access Helper

OpenAlex Citation Counts

OpenAlex Citations Logo

OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Quantitative analysis and clinico-pathological correlations of different dipeptide repeat protein pathologies in C9ORF72 mutation carriers
Ian R. Mackenzie, Petra Frick, Friedrich A. Grässer, et al.
Acta Neuropathologica (2015) Vol. 130, Iss. 6, pp. 845-861
Closed Access | Times Cited: 219

Showing 1-25 of 219 citing articles:

Decoding ALS: from genes to mechanism
J. Paul Taylor, Robert H. Brown, Don W. Cleveland
Nature (2016) Vol. 539, Iss. 7628, pp. 197-206
Open Access | Times Cited: 1821
C9orf72-mediated ALS and FTD: multiple pathways to disease
Rubika Balendra, Adrian M. Isaacs
Nature Reviews Neurology (2018) Vol. 14, Iss. 9, pp. 544-558
Open Access | Times Cited: 616
Haploinsufficiency leads to neurodegeneration in C9ORF72 ALS/FTD human induced motor neurons
Yingxiao Shi, Shao‐Yu Lin, Kim A. Staats, et al.
Nature Medicine (2018) Vol. 24, Iss. 3, pp. 313-325
Open Access | Times Cited: 519
Gain of Toxicity from ALS/FTD-Linked Repeat Expansions in C9ORF72 Is Alleviated by Antisense Oligonucleotides Targeting GGGGCC-Containing RNAs
Jie Jiang, Qiang Zhu, Tania F. Gendron, et al.
Neuron (2016) Vol. 90, Iss. 3, pp. 535-550
Open Access | Times Cited: 502
In Situ Structure of Neuronal C9orf72 Poly-GA Aggregates Reveals Proteasome Recruitment
Qiang Guo, Carina Lehmer, Antonio Martínez-Sánchez, et al.
Cell (2018) Vol. 172, Iss. 4, pp. 696-705.e12
Open Access | Times Cited: 379
Molecular neuropathology of frontotemporal dementia: insights into disease mechanisms from postmortem studies
Ian R. Mackenzie, Manuela Neumann
Journal of Neurochemistry (2016) Vol. 138, Iss. S1, pp. 54-70
Open Access | Times Cited: 315
C9ORF72 poly(GA) aggregates sequester and impair HR23 and nucleocytoplasmic transport proteins
Yong‐Jie Zhang, Tania F. Gendron, Jonathan C. Grima, et al.
Nature Neuroscience (2016) Vol. 19, Iss. 5, pp. 668-677
Open Access | Times Cited: 300
ALS Genes in the Genomic Era and their Implications for FTD
Hung Phuoc Nguyen, Christine Van Broeckhoven, Julie van der Zee
Trends in Genetics (2018) Vol. 34, Iss. 6, pp. 404-423
Open Access | Times Cited: 272
Molecular mechanisms underlying nucleotide repeat expansion disorders
Indranil Malik, Chase P. Kelley, Eric T. Wang, et al.
Nature Reviews Molecular Cell Biology (2021) Vol. 22, Iss. 9, pp. 589-607
Open Access | Times Cited: 263
CRISPR–Cas9 screens in human cells and primary neurons identify modifiers of C9ORF72 dipeptide-repeat-protein toxicity
Nicholas J. Kramer, Michael S. Haney, David W. Morgens, et al.
Nature Genetics (2018) Vol. 50, Iss. 4, pp. 603-612
Open Access | Times Cited: 222
Translation of Expanded CGG Repeats into FMRpolyG Is Pathogenic and May Contribute to Fragile X Tremor Ataxia Syndrome
Chantal Sellier, Ronald A.M. Buijsen, Fang He, et al.
Neuron (2017) Vol. 93, Iss. 2, pp. 331-347
Open Access | Times Cited: 215
RAN translation at C9orf72-associated repeat expansions is selectively enhanced by the integrated stress response
Katelyn M. Green, M. Rebecca Glineburg, Michael G. Kearse, et al.
Nature Communications (2017) Vol. 8, Iss. 1
Open Access | Times Cited: 209
Heterochromatin anomalies and double-stranded RNA accumulation underlie C9orf72 poly(PR) toxicity
Yong‐Jie Zhang, Lin Guo, Patrick Gonzales, et al.
Science (2019) Vol. 363, Iss. 6428
Open Access | Times Cited: 208
C9ORF72-ALS/FTD-associated poly(GR) binds Atp5a1 and compromises mitochondrial function in vivo
So Yoen Choi, Rodrigo López‐González, Gopinath Krishnan, et al.
Nature Neuroscience (2019) Vol. 22, Iss. 6, pp. 851-862
Open Access | Times Cited: 202
Reduced C9ORF72 function exacerbates gain of toxicity from ALS/FTD-causing repeat expansion in C9orf72
Qiang Zhu, Jie Jiang, Tania F. Gendron, et al.
Nature Neuroscience (2020) Vol. 23, Iss. 5, pp. 615-624
Open Access | Times Cited: 197
Sense-encoded poly-GR dipeptide repeat proteins correlate to neurodegeneration and uniquely co-localize with TDP-43 in dendrites of repeat-expanded C9orf72 amyotrophic lateral sclerosis
Shahram Saberi, Jennifer E. Stauffer, Jie Jiang, et al.
Acta Neuropathologica (2017) Vol. 135, Iss. 3, pp. 459-474
Open Access | Times Cited: 174
C9orf72 poly(GR) aggregation induces TDP-43 proteinopathy
Casey Cook, Yanwei Wu, Hana M. Odeh, et al.
Science Translational Medicine (2020) Vol. 12, Iss. 559
Open Access | Times Cited: 161
RNA toxicity in non‐coding repeat expansion disorders
Bart Swinnen, Wim Robberecht, Ludo Van Den Bosch
The EMBO Journal (2019) Vol. 39, Iss. 1
Open Access | Times Cited: 147
p53 is a central regulator driving neurodegeneration caused by C9orf72 poly(PR)
Maya Maor-Nof, Zohar Shipony, Rodrigo López‐González, et al.
Cell (2021) Vol. 184, Iss. 3, pp. 689-708.e20
Open Access | Times Cited: 141
Autophagy and ALS: mechanistic insights and therapeutic implications
Jason P. Chua, Hortense de Calbiac, Edor Kabashi, et al.
Autophagy (2021) Vol. 18, Iss. 2, pp. 254-282
Open Access | Times Cited: 118
Non-neuronal cells in amyotrophic lateral sclerosis — from pathogenesis to biomarkers
Björn F. Vahsen, Elizabeth Gray, Alexander G. Thompson, et al.
Nature Reviews Neurology (2021) Vol. 17, Iss. 6, pp. 333-348
Open Access | Times Cited: 117
C9ORF72: What It Is, What It Does, and Why It Matters
Julie Smeyers, Elena-Gaia Banchi, Morwena Latouche
Frontiers in Cellular Neuroscience (2021) Vol. 15
Open Access | Times Cited: 113
The sense of antisense therapies in ALS
Sien Hilde Van Daele, Pegah Masrori, Philip Van Damme, et al.
Trends in Molecular Medicine (2024) Vol. 30, Iss. 3, pp. 252-262
Open Access | Times Cited: 24
Amyotrophic lateral sclerosis caused by hexanucleotide repeat expansions in C9orf72: from genetics to therapeutics
Sarah Mizielinska, Guillaume M. Hautbergue, Tania F. Gendron, et al.
The Lancet Neurology (2025) Vol. 24, Iss. 3, pp. 261-274
Closed Access | Times Cited: 3
Dysregulated molecular pathways in amyotrophic lateral sclerosis–frontotemporal dementia spectrum disorder
Fen‐Biao Gao, Sandra Almeida, Rodrigo López‐González
The EMBO Journal (2017) Vol. 36, Iss. 20, pp. 2931-2950
Open Access | Times Cited: 166
Page 1 - Next Page

Scroll to top