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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Loss of CC2D1A in Glutamatergic Neurons Results in Autistic-Like Features in Mice
Cheng-Yi Yang, Yu-Chieh Hung, Kuan‐Hsiang Cheng, et al.
Neurotherapeutics (2021) Vol. 18, Iss. 3, pp. 2021-2039
Open Access | Times Cited: 10

Showing 10 citing articles:

Oxytocin signaling in the ventral tegmental area mediates social isolation-induced craving for social interaction
Hsin-Tzu Chang, Kuan‐Hsiang Cheng, Yu-Chieh Hung, et al.
Journal of Biomedical Science (2025) Vol. 32, Iss. 1
Open Access
eEF2 in the prefrontal cortex promotes excitatory synaptic transmission and social novelty behavior
Xuanyue Ma, Liuren Li, Ziming Li, et al.
EMBO Reports (2022) Vol. 23, Iss. 10
Open Access | Times Cited: 14
CC2D1A causes ciliopathy, intellectual disability, heterotaxy, renal dysplasia, and abnormal CSF flow
A. Kim, Irmak Sakin, Stephen Viviano, et al.
Life Science Alliance (2024) Vol. 7, Iss. 10, pp. e202402708-e202402708
Open Access | Times Cited: 2
Metalomics Revealed that Changes of Serum Elements were Associated with Oxidative Stress-Induced Inflammation of Cortex in a Mouse Model of Autism
Can Cao, Li J, Wenqi Cui, et al.
Biological Trace Element Research (2024)
Closed Access | Times Cited: 2
Increased propensity for infantile spasms and altered neocortical excitation-inhibition balance in a mouse model of down syndrome carrying human chromosome 21
Li‐Rong Shao, Feng Gao, Viveka Chinnasamy, et al.
Neurobiology of Disease (2023) Vol. 184, pp. 106198-106198
Open Access | Times Cited: 4
A nonsense CC2D1A variant is associated with congenital anomalies, motor delay, hypotonia, and slight deformities
Sheng Yi, Xianglian Tang, Qiang Zhang, et al.
Heliyon (2024) Vol. 10, Iss. 6, pp. e27946-e27946
Open Access | Times Cited: 1
Endosomes serve as signaling platforms for RIG-I ubiquitination and activation
Kuan‐Ru Chen, Chia-Yu Yang, S Shu, et al.
Science Advances (2024) Vol. 10, Iss. 45
Open Access | Times Cited: 1
Loss of oxytocin receptors in hilar mossy cells impairs social discrimination
Yu-Chieh Hung, Yi‐Jen Wu, Miao-Er Chien, et al.
Neurobiology of Disease (2023) Vol. 187, pp. 106311-106311
Open Access | Times Cited: 3
High-Fat Diet Exacerbates Autistic-Like Restricted Repetitive Behaviors and Social Abnormalities in CC2D1A Conditional Knockout Mice
Yu-Chiao Wang, Chin‐Hao Chen, Cheng-Yi Yang, et al.
Molecular Neurobiology (2022) Vol. 60, Iss. 3, pp. 1331-1352
Closed Access | Times Cited: 4
Oxytocin treatment rescues irritability-like behavior in Cc2d1a conditional knockout mice
Kuan‐Hsiang Cheng, Yu-Chieh Hung, Pin Ling, et al.
Neuropsychopharmacology (2024) Vol. 49, Iss. 11, pp. 1792-1802
Closed Access
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