OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Inhibition of xanthine oxidase alleviated pancreatic necrosis via HIF-1α-regulated LDHA and NLRP3 signaling pathway in acute pancreatitis
Juan Rong, Chenxia Han, Yan Huang, et al.
Acta Pharmaceutica Sinica B (2024) Vol. 14, Iss. 8, pp. 3591-3604
Open Access | Times Cited: 8

Showing 8 citing articles:

Unveiling the Emerging Role of Xanthine Oxidase in Acute Pancreatitis: Beyond Reactive Oxygen Species
Chenxia Han, Yaling Wu, Juan Rong, et al.
Antioxidants (2025) Vol. 14, Iss. 1, pp. 95-95
Open Access

HIF-1α Enhances Intestinal Injury and Inflammation in Severe Acute Pancreatitis Through NLRP3 Inflammasome Activation
Tao Gao, Hao Zhang, Yuan Xu, et al.
Digestive Diseases and Sciences (2025)
Closed Access

Exosomes in acute pancreatitis: Pathways to cellular death regulation and clinical application potential
Kaiyi Ruan, Jinglei Zhang, Zhaowei Chu, et al.
International Immunopharmacology (2025) Vol. 153, pp. 114491-114491
Closed Access

Pancreatic Morphology, Immunology, and the Pathogenesis of Acute Pancreatitis
Tudorel Mihoc, Silviu Constantin Latcu, Cosmin-Ciprian Secasan, et al.
Biomedicines (2024) Vol. 12, Iss. 11, pp. 2627-2627
Open Access | Times Cited: 1

Reactive oxygen species and oxidative stress in acute pancreatitis: Pathogenesis and new therapeutic interventions
Chuanchao Xia, Hong‐Tan Chen, Hao Deng, et al.
World Journal of Gastroenterology (2024) Vol. 30, Iss. 45, pp. 4771-4780
Closed Access

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