OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

The impact of murine LRRK2 G2019S transgene overexpression on acute responses to inflammatory challenge
Darcy Litteljohn, Chris Rudyk, Zach Dwyer, et al.
Brain Behavior and Immunity (2017) Vol. 67, pp. 246-256
Closed Access | Times Cited: 22

Showing 22 citing articles:

Lrrk2 alleles modulate inflammation during microbial infection of mice in a sex-dependent manner
Bojan Shutinoski, Mansoureh Hakimi, Irene E. Harmsen, et al.
Science Translational Medicine (2019) Vol. 11, Iss. 511
Closed Access | Times Cited: 83

G2019S LRRK2 mutation facilitates α-synuclein neuropathology in aged mice
Salvatore Novello, Ludovico Arcuri, Sandra Doveró, et al.
Neurobiology of Disease (2018) Vol. 120, pp. 21-33
Open Access | Times Cited: 62

Leucine Rich Repeat Kinase 2 and Innate Immunity
Diba Ahmadi Rastegar, Nicolas Dzamko
Frontiers in Neuroscience (2020) Vol. 14
Open Access | Times Cited: 55

Effect of LRRK2 protein and activity on stimulated cytokines in human monocytes and macrophages
Diba Ahmadi Rastegar, Laura P. Hughes, Gayathri Perera, et al.
npj Parkinson s Disease (2022) Vol. 8, Iss. 1
Open Access | Times Cited: 30

Leucine-rich repeat kinase-2 (LRRK2) modulates microglial phenotype and dopaminergic neurodegeneration
Zach Dwyer, Chris Rudyk, Ashley Thompson, et al.
Neurobiology of Aging (2020) Vol. 91, pp. 45-55
Open Access | Times Cited: 39

Monoamine signaling and neuroinflammation: mechanistic connections and implications for neuropsychiatric disorders
Shalini Saggu, Andrew Pless, Emily Dew, et al.
Frontiers in Immunology (2025) Vol. 16
Open Access

Leucine-rich repeat kinase-2 (LRRK2) modulates paraquat-induced inflammatory sickness and stress phenotype
Chris Rudyk, Zach Dwyer, Shawn Hayley
Journal of Neuroinflammation (2019) Vol. 16, Iss. 1
Open Access | Times Cited: 28

G2019S Variation in LRRK2: An Ideal Model for the Study of Parkinson’s Disease?
Chao Ren, Yu Ding, Shi‐Zhuang Wei, et al.
Frontiers in Human Neuroscience (2019) Vol. 13
Open Access | Times Cited: 27

Physiological and pathological functions of LRRK2: implications from substrate proteins
Miho Araki, Genta Ito, Taisuke Tomita
Neuronal Signaling (2018) Vol. 2, Iss. 4
Open Access | Times Cited: 24

Age-related pathology after adenoviral overexpression of the leucine-rich repeat kinase 2 in the mouse striatum
Astrid Kritzinger, Boris Ferger, Frank Gillardon, et al.
Neurobiology of Aging (2018) Vol. 66, pp. 97-111
Open Access | Times Cited: 17

Neuropsychiatric consequences of childhood group A streptococcal infection: A systematic review of preclinical models
Santiago Mora, Elena Martín-González, Pilar Flores, et al.
Brain Behavior and Immunity (2019) Vol. 86, pp. 53-62
Closed Access | Times Cited: 16

Increased Compulsivity in Adulthood after Early Adolescence Immune Activation: Preclinical Evidence
Santiago Mora, Elena Martín-González, Ángeles Prados-Pardo, et al.
International Journal of Environmental Research and Public Health (2021) Vol. 18, Iss. 9, pp. 4684-4684
Open Access | Times Cited: 5

References

Immune system (2024), pp. 471-578
Closed Access

Inflammatory Roads to Parkinson’s Disease
Hymie Anisman, Shawn Hayley, Alexander Kusnecov
Immune system (2024), pp. 393-418
Closed Access

LRRK2 G2019S and Parkinson’s disease: insight from Neuroinflammation
Xiao-Yan Yao, Lina Guan, Qi Chen, et al.
Postgraduate Medical Journal (2023) Vol. 100, Iss. 1179, pp. 4-11
Closed Access | Times Cited: 1

References

Elsevier eBooks (2018), pp. 555-664
Closed Access

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