OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

PlexinA4-Semaphorin3A-mediated crosstalk between main cortical interneuron classes is required for superficial interneuron lamination
Greta Limoni, Sahana Murthy, Denis Jabaudon, et al.
Cell Reports (2021) Vol. 34, Iss. 4, pp. 108644-108644
Open Access | Times Cited: 14

Showing 14 citing articles:

Semaphorins and Plexins in central nervous system patterning: the key to it all?
Greta Limoni, Mathieu Niquille
Current Opinion in Neurobiology (2021) Vol. 66, pp. 224-232
Open Access | Times Cited: 53

Interneuron odyssey: molecular mechanisms of tangential migration
Ikram Toudji, Asmaa Toumi, Émile Chamberland, et al.
Frontiers in Neural Circuits (2023) Vol. 17
Open Access | Times Cited: 11

A high-definition spatiotemporal transcriptomic atlas of mammalian kidney development
Shan Jiang, Hao Yu, Ting C. Zhao, et al.
The Innovation (2025), pp. 100767-100767
Open Access

Biological Insights from Schizophrenia-associated Loci in Ancestral Populations
Tim B. Bigdeli, Chris Chatzinakos, Jaroslav Bendl, et al.
medRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access | Times Cited: 3

Toward a probabilistic definition of neural cell types
Mahéva Andriatsilavo, Bassem A. Hassan
Current Opinion in Neurobiology (2025) Vol. 92, pp. 103035-103035
Open Access

Whole Genome Sequencing Analysis to Identify Candidate Genes Associated With the rib eye Muscle Area in Hu Sheep
Yuan Zhao, Xiaoxue Zhang, Fadi Li, et al.
Frontiers in Genetics (2022) Vol. 13
Open Access | Times Cited: 16

Semaphorin 3A influences neuronal processes that are altered in patients with autism spectrum disorder: Potential diagnostic and therapeutic implications
Carmela Matrone, Gabriella Ferretti
Neuroscience & Biobehavioral Reviews (2023) Vol. 153, pp. 105338-105338
Closed Access | Times Cited: 7

Cell type specific roles of FOXP1 during early neocortical murine development
Ana Ortiz, Fatma Ayhan, Matthew J. Harper, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access | Times Cited: 2

Modelling and Refining Neuronal Circuits with Guidance Cues: Involvement of Semaphorins
Greta Limoni
International Journal of Molecular Sciences (2021) Vol. 22, Iss. 11, pp. 6111-6111
Open Access | Times Cited: 15

DOT1L deletion impairs the development of cortical parvalbumin-expressing interneurons
Arquimedes Cheffer, Marta Garcia‐Miralles, Esther Maier, et al.
Cerebral Cortex (2023) Vol. 33, Iss. 19, pp. 10272-10285
Open Access | Times Cited: 6

PlexinA1-deficient mice exhibit decreased cell density and augmented oxidative stress in parvalbumin-expressing interneurons in the medial prefrontal cortex
Mst Sharifa Jahan, Takamasa Tsuzuki, Takuji Ito, et al.
IBRO Neuroscience Reports (2022) Vol. 13, pp. 500-512
Open Access | Times Cited: 7

Cxcr4 and Ackr3 regulate allocation of caudal ganglionic eminence-derived interneurons to superficial cortical layers
Sathish Venkataramanappa, Friederike Saaber, Philipp Abe, et al.
Cell Reports (2022) Vol. 40, Iss. 5, pp. 111157-111157
Open Access | Times Cited: 6

In the developing cerebral cortex: axonogenesis, synapse formation, and synaptic plasticity are regulated by SATB2 target genes
Qiufang Guo, Yaqiong Wang, Qing Wang, et al.
Pediatric Research (2022) Vol. 93, Iss. 6, pp. 1519-1527
Closed Access | Times Cited: 6

DOT1L deletion impairs the development of cortical Parvalbumin-expressing interneurons
Arquimedes Cheffer, Marta Garcia‐Miralles, Esther Maier, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2023)
Open Access

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