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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Understanding the contributions of VPS35 and the retromer in neurodegenerative disease
Erin T. Williams, Xi Chen, P. Anthony Otero, et al.
Neurobiology of Disease (2022) Vol. 170, pp. 105768-105768
Open Access | Times Cited: 31

Showing 1-25 of 31 citing articles:

Mitochondrial dysfunction in Parkinson’s disease – a key disease hallmark with therapeutic potential
Martin T. Henrich, Wolfgang H. Oertel, D. James Surmeier, et al.
Molecular Neurodegeneration (2023) Vol. 18, Iss. 1
Open Access | Times Cited: 94
CD63 sorts cholesterol into endosomes for storage and distribution via exosomes
Roberta Palmulli, Mickaël Couty, Melissa C. Piontek, et al.
Nature Cell Biology (2024) Vol. 26, Iss. 7, pp. 1093-1109
Open Access | Times Cited: 17
DNA Damage and Parkinson’s Disease
Gerd P. Pfeifer
International Journal of Molecular Sciences (2024) Vol. 25, Iss. 8, pp. 4187-4187
Open Access | Times Cited: 8
VPS35 and retromer dysfunction in Parkinson's disease
Jordan Rowlands, Darren J. Moore
Philosophical Transactions of the Royal Society B Biological Sciences (2024) Vol. 379, Iss. 1899
Open Access | Times Cited: 5
α-Synuclein: Multiple pathogenic roles in trafficking and proteostasis pathways in Parkinson’s disease
Annie J. Zalon, Drew J. Quiriconi, Caleb Pitcairn, et al.
The Neuroscientist (2024) Vol. 30, Iss. 5, pp. 612-635
Closed Access | Times Cited: 5
Genetic Evidence for Endolysosomal Dysfunction in Parkinson’s Disease: A Critical Overview
Vidal Yahya, Alessio Di Fonzo, Edoardo Monfrini
International Journal of Molecular Sciences (2023) Vol. 24, Iss. 7, pp. 6338-6338
Open Access | Times Cited: 12
The association of GNB5 with Alzheimer disease revealed by genomic analysis restricted to variants impacting gene function
Jianhua Zhang, Mritunjay Pandey, Adam M. Awe, et al.
The American Journal of Human Genetics (2024) Vol. 111, Iss. 3, pp. 473-486
Open Access | Times Cited: 4
Probe-dependent Proximity Profiling (ProPPr) Uncovers Similarities and Differences in Phospho-Tau-Associated Proteomes Between Tauopathies
Dmytro Morderer, Melissa C. Wren, Feilin Liu, et al.
Molecular Neurodegeneration (2025) Vol. 20, Iss. 1
Open Access
Bioinformatics analysis of oxidative phosphorylation-related differentially expressed genes in osteoporosis
Songmao Wang, Yaling Wang, Minfeng Gan, et al.
European journal of medical research (2025) Vol. 30, Iss. 1
Open Access
Inhibition of LRRK2 kinase activity rescues deficits in striatal dopamine physiology in VPS35 p.D620N knock-in mice
Mengfei Bu, Jordan Follett, Isaac Deng, et al.
npj Parkinson s Disease (2023) Vol. 9, Iss. 1
Open Access | Times Cited: 8
Mutant mice with rod-specific VPS35 deletion exhibit retinal α-synuclein pathology-associated degeneration
Cheng Fu, Nan Yang, Jen-Zen Chuang, et al.
Nature Communications (2024) Vol. 15, Iss. 1
Open Access | Times Cited: 2
The function of Golgi apparatus in LRRK2-associated Parkinson’s disease
Yonghang Wei, Maher Un Nisa Awan, Li‐Ping Bai, et al.
Frontiers in Molecular Neuroscience (2023) Vol. 16
Open Access | Times Cited: 5
Novel variants in genes related to vesicle-mediated-transport modify Parkinson's disease risk
Orly Goldstein, Mali Gana‐Weisz, Sandro Banfi, et al.
Molecular Genetics and Metabolism (2023) Vol. 139, Iss. 2, pp. 107608-107608
Closed Access | Times Cited: 5
VPS35 and α-Synuclein fail to interact to modulate neurodegeneration in rodent models of Parkinson’s disease
Xi Chen, Elpida Tsika, Nathan Levine, et al.
Molecular Neurodegeneration (2023) Vol. 18, Iss. 1
Open Access | Times Cited: 5
VPS35 or retromer as a potential target for neurodegenerative disorders: barriers to progress
Anika Wu, Daehoon Lee, Wen-Cheng Xiong
Expert Opinion on Therapeutic Targets (2024) Vol. 28, Iss. 8, pp. 701-712
Open Access | Times Cited: 1
The crucial role of VPS35 and SHH in Parkinson’s disease: Understanding the mechanisms behind the neurodegenerative disorder
Kritika Bhardwaj, Akanksha Jha, Abhishek Roy, et al.
Brain Research (2024) Vol. 1845, pp. 149204-149204
Closed Access | Times Cited: 1
Distinct regulation of Tau Monomer and aggregate uptake and intracellular accumulation in human neurons
Amir Tayaranian Marvian, Tabea Strauß, Qilin Tang, et al.
Molecular Neurodegeneration (2024) Vol. 19, Iss. 1
Open Access | Times Cited: 1
Aging, Parkinson’s Disease, and Models: What Are the Challenges?
Ilaria Bellantuono, Heather Mortiboys, Emily M. Rocha, et al.
(2023) Vol. 1, Iss. 1, pp. 20230010-20230010
Open Access | Times Cited: 3
Probe-dependent Proximity Profiling (ProPPr) Uncovers Similarities and Differences in Phospho-Tau-Associated Proteomes Between Tauopathies
Dmytro Morderer, Melissa C. Wren, Feilin Liu, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access
Protrudin acts at ER-endosome contacts to promote KIF5-mediated endosomal fission and endosome-to-Golgi transport
Julia Kleniuk, Aishwarya G. Nadadhur, Emily Wolfenden, et al.
(2024)
Open Access
Exploring the triad: VPS35, neurogenesis, and neurodegenerative diseases
Zixiong Qiu, Xuliang Deng, Yuan Fu, et al.
Journal of Neurochemistry (2024)
Closed Access
Mechanisms of lysosomal tubulation and sorting driven by LRRK2
Luis Bonet‐Ponce, Jillian H. Kluss, Mark Cookson
Biochemical Society Transactions (2024) Vol. 52, Iss. 4, pp. 1909-1919
Closed Access
Impaired neurogenesis and synaptogenesis in iPSC-derived Parkinson’s patient cortical neurons with D620N VPS35 mutation
Ute Scheller, ChoongKu Lee, Philip Seibler, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access
A human neuronal model of sporadic Alzheimer's disease induced by FBXO2 downregulation shows A β aggregation, tau hyperphosphorylation and functional network impairment
Alicia González Díaz, Andrea Possenti, Gustavo Antonio Urrutia, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access
Phenolic-enabled nanotechnology: a new strategy for central nervous system disease therapy
Yuyi Zheng, Xiaojie Chen, Yi Wang, et al.
Journal of Zhejiang University SCIENCE B (2024) Vol. 25, Iss. 10, pp. 890-913
Closed Access
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