OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

C9orf72 BAC Transgenic Mice Display Typical Pathologic Features of ALS/FTD
Jacqueline G. O’Rourke, Laurent Bogdanik, A.K.M. Ghulam Muhammad, et al.
Neuron (2015) Vol. 88, Iss. 5, pp. 892-901
Open Access | Times Cited: 284

Showing 1-25 of 284 citing articles:

Decoding ALS: from genes to mechanism
J. Paul Taylor, Robert H. Brown, Don W. Cleveland
Nature (2016) Vol. 539, Iss. 7628, pp. 197-206
Open Access | Times Cited: 1821

Amyotrophic Lateral Sclerosis
Robert H. Brown, Ammar Al‐Chalabi
New England Journal of Medicine (2017) Vol. 377, Iss. 2, pp. 162-172
Open Access | Times Cited: 1704

Microglia in neurodegeneration
Suzanne E. Hickman, Saef Izzy, Pritha Sen, et al.
Nature Neuroscience (2018) Vol. 21, Iss. 10, pp. 1359-1369
Open Access | Times Cited: 1389

ALS Genetics, Mechanisms, and Therapeutics: Where Are We Now?
Rita Mejzini, Loren L. Flynn, Ianthe Pitout, et al.
Frontiers in Neuroscience (2019) Vol. 13
Open Access | Times Cited: 658

C9orf72-mediated ALS and FTD: multiple pathways to disease
Rubika Balendra, Adrian M. Isaacs
Nature Reviews Neurology (2018) Vol. 14, Iss. 9, pp. 544-558
Open Access | Times Cited: 616

Haploinsufficiency leads to neurodegeneration in C9ORF72 ALS/FTD human induced motor neurons
Yingxiao Shi, Shao‐Yu Lin, Kim A. Staats, et al.
Nature Medicine (2018) Vol. 24, Iss. 3, pp. 313-325
Open Access | Times Cited: 519

Gain of Toxicity from ALS/FTD-Linked Repeat Expansions in C9ORF72 Is Alleviated by Antisense Oligonucleotides Targeting GGGGCC-Containing RNAs
Jie Jiang, Qiang Zhu, Tania F. Gendron, et al.
Neuron (2016) Vol. 90, Iss. 3, pp. 535-550
Open Access | Times Cited: 502

Animal models of neurodegenerative diseases
Ted M. Dawson, Todd E. Golde, Clotilde Lagier‐Tourenne
Nature Neuroscience (2018) Vol. 21, Iss. 10, pp. 1370-1379
Open Access | Times Cited: 491

Poly(GR) in C9ORF72 -Related ALS/FTD Compromises Mitochondrial Function and Increases Oxidative Stress and DNA Damage in iPSC-Derived Motor Neurons
Rodrigo López‐González, Yubing Lu, Tania F. Gendron, et al.
Neuron (2016) Vol. 92, Iss. 2, pp. 383-391
Open Access | Times Cited: 384

Loss of C9 ORF 72 impairs autophagy and synergizes with polyQ Ataxin‐2 to induce motor neuron dysfunction and cell death
Chantal Sellier, Maria‐Letizia Campanari, Camille Corbier, et al.
The EMBO Journal (2016) Vol. 35, Iss. 12, pp. 1276-1297
Open Access | Times Cited: 366

C9orf72 BAC Mouse Model with Motor Deficits and Neurodegenerative Features of ALS/FTD
Yuanjing Liu, Amrutha Pattamatta, Tao Zu, et al.
Neuron (2016) Vol. 90, Iss. 3, pp. 521-534
Open Access | Times Cited: 344

Molecular neuropathology of frontotemporal dementia: insights into disease mechanisms from postmortem studies
Ian R. Mackenzie, Manuela Neumann
Journal of Neurochemistry (2016) Vol. 138, Iss. S1, pp. 54-70
Open Access | Times Cited: 315

The ALS/FTLD associated protein C9orf72 associates with SMCR8 and WDR41 to regulate the autophagy-lysosome pathway
Peter M. Sullivan, Xiaolai Zhou, Adam M. Robins, et al.
Acta Neuropathologica Communications (2016) Vol. 4, Iss. 1
Open Access | Times Cited: 270

Poly(GP) proteins are a useful pharmacodynamic marker for C9ORF72 -associated amyotrophic lateral sclerosis
Tania F. Gendron, Jeannie Chew, Jeannette N. Stankowski, et al.
Science Translational Medicine (2017) Vol. 9, Iss. 383
Open Access | Times Cited: 253

Human C9ORF72 Hexanucleotide Expansion Reproduces RNA Foci and Dipeptide Repeat Proteins but Not Neurodegeneration in BAC Transgenic Mice
Owen M. Peters, Gabriela Toro Cabrera, Hélène Tran, et al.
Neuron (2015) Vol. 88, Iss. 5, pp. 902-909
Open Access | Times Cited: 244

The Role of Dipeptide Repeats in C9ORF72-Related ALS-FTD
Brian D. Freibaum, J. Paul Taylor
Frontiers in Molecular Neuroscience (2017) Vol. 10
Open Access | Times Cited: 244

Genetics of Amyotrophic Lateral Sclerosis
Mehdi Ghasemi, Robert H. Brown
Cold Spring Harbor Perspectives in Medicine (2017) Vol. 8, Iss. 5, pp. a024125-a024125
Open Access | Times Cited: 222

The Overlapping Genetics of Amyotrophic Lateral Sclerosis and Frontotemporal Dementia
Yevgeniya Abramzon, Pietro Fratta, Bryan J. Traynor, et al.
Frontiers in Neuroscience (2020) Vol. 14
Open Access | Times Cited: 203

The expanding biology of the C9orf72 nucleotide repeat expansion in neurodegenerative disease
Aaron R. Haeusler, Christopher J. Donnelly, Jeffrey D. Rothstein
Nature reviews. Neuroscience (2016) Vol. 17, Iss. 6, pp. 383-395
Open Access | Times Cited: 183

ALS: A bucket of genes, environment, metabolism and unknown ingredients
Mónica Zufiría, Francisco J. Gil‐Bea, Roberto Fernández‐Torrón, et al.
Progress in Neurobiology (2016) Vol. 142, pp. 104-129
Closed Access | Times Cited: 183

C9orf72 binds SMCR8, localizes to lysosomes, and regulates mTORC1 signaling
Joseph Amick, Agnes Roczniak-Ferguson, Shawn M. Ferguson
Molecular Biology of the Cell (2016) Vol. 27, Iss. 20, pp. 3040-3051
Open Access | Times Cited: 175

Loss of C9orf72 Enhances Autophagic Activity via Deregulated mTOR and TFEB Signaling
Janet Ugolino, Yon Ju Ji, Karen Conchina, et al.
PLoS Genetics (2016) Vol. 12, Iss. 11, pp. e1006443-e1006443
Open Access | Times Cited: 173

RNA Dysregulation in Amyotrophic Lateral Sclerosis
Zoé Butti, Scott B. Patten
Frontiers in Genetics (2019) Vol. 9
Open Access | Times Cited: 166

Analysis of short tandem repeat expansions and their methylation state with nanopore sequencing
Pay Gießelmann, Björn Brändl, Etienne Raimondeau, et al.
Nature Biotechnology (2019) Vol. 37, Iss. 12, pp. 1478-1481
Open Access | Times Cited: 166

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