OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Orexin 1 and 2 Receptors in the Prelimbic Cortex Modulate Threat Valuation
Victor P. M. N. Soares, Telma Gonçalves Carneiro Spera de Andrade, Newton S. Canteras, et al.
Neuroscience (2021) Vol. 468, pp. 158-167
Closed Access | Times Cited: 7

Showing 7 citing articles:

Orexin 1 Receptor Antagonism in the Basolateral Amygdala Shifts the Balance From Pro- to Antistress Signaling and Behavior
Jazmine D.W. Yaeger, Kevin T. Krupp, Benjamin Jacobs, et al.
Biological Psychiatry (2022) Vol. 91, Iss. 9, pp. 841-852
Open Access | Times Cited: 22

Spatial mRNA expression patterns of orexin receptors in the dorsal hippocampus
Gina Marie Krause, Lara Mariel Chirich Barreira, Anne Albrecht
Scientific Reports (2024) Vol. 14, Iss. 1
Open Access | Times Cited: 1

The role of the orexin system in the neurobiology of anxiety disorders: Potential for a novel treatment target
Harry Fagan, Nathan T. M. Huneke, Katharina Domschke, et al.
Neuroscience Applied (2023) Vol. 3, pp. 103922-103922
Open Access | Times Cited: 3

Spatial mRNA expression patterns of orexin receptors in the dorsal hippocampus
Gina Marie Krause, Lara Mariel Chirich Barreira, Anne Albrecht
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access

Orexin mechanisms in the prelimbic cortex modulate the expression of contextual conditioned fear
Gabriela V. M. Oliveira, Paloma Molina Hernandes, Fábio H. dos Santos, et al.
Psychopharmacology (2024)
Closed Access

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