OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Maternal fructose consumption downregulates hippocampal catalase expression via DNA methylation in rat offspring
Genki Mizuno, Eiji Munetsuna, Hiroya Yamada, et al.
Nutrition Research (2021) Vol. 92, pp. 40-48
Closed Access | Times Cited: 15

Showing 15 citing articles:

Low mitochondrial DNA copy number in peripheral blood mononuclear cells is associated with future mortality risk: a long-term follow-up study from Japan
Genki Mizuno, Hiroya Yamada, Yoshiki Tsuboi, et al.
The journal of nutrition health & aging (2024) Vol. 28, Iss. 1, pp. 100013-100013
Open Access | Times Cited: 5

Differential effects of excess high-fructose corn syrup on the DNA methylation of hippocampal neurotrophic factor in childhood and adolescence
Itsuki Kageyama, Hiroya Yamada, Eiji Munetsuna, et al.
PLoS ONE (2022) Vol. 17, Iss. 6, pp. e0270144-e0270144
Open Access | Times Cited: 13

Maternal Fructose Diet-Induced Developmental Programming
Michael D. Thompson, Brian J. DeBosch
Nutrients (2021) Vol. 13, Iss. 9, pp. 3278-3278
Open Access | Times Cited: 13

Pathophysiological mechanisms of hypertension development induced by fructose consumption
Chuanming Xu, Jun Yu
Food & Function (2022) Vol. 13, Iss. 4, pp. 1702-1717
Closed Access | Times Cited: 10

Impact of maternal fructose intake on liver stem/progenitor cells in offspring: Insights into developmental origins of health and disease
Yoshitaka Ando, Eiji Munetsuna, Hiroya Yamada, et al.
Life Sciences (2023) Vol. 336, pp. 122315-122315
Closed Access | Times Cited: 5

Developmental Programming in Animal Models: Critical Evidence of Current Environmental Negative Changes
Victoria Ramírez, Regina J. Bautista, Oswaldo Frausto-González, et al.
Reproductive Sciences (2022) Vol. 30, Iss. 2, pp. 442-463
Open Access | Times Cited: 9

Maternal fructose intake predisposes rat offspring to metabolic disorders via abnormal hepatic programming
Eiji Munetsuna, Hiroya Yamada, Mirai Yamazaki, et al.
The FASEB Journal (2021) Vol. 35, Iss. 12
Closed Access | Times Cited: 11

Lipid peroxidation and neuroinflammation: A possible link between maternal fructose intake and delay of acquisition of neonatal reflexes in Wistar female rats
Facundo H. Prado Spalm, Marié L. Cuervo Sánchez, Natalia E. Furland, et al.
Developmental Neurobiology (2023) Vol. 83, Iss. 5-6, pp. 167-183
Open Access | Times Cited: 4

Effects of High-Fructose Corn Syrup Intake on Glucocorticoid Metabolism in Rats During Childhood, Adolescence and Adulthood
Yuki Nouchi, Eiji Munetsuna, Hiroya Yamada, et al.
Experimental and Clinical Endocrinology & Diabetes (2022) Vol. 130, Iss. 12, pp. 814-820
Open Access | Times Cited: 3

High-fructose corn syrup intake increases hepatic mitochondrial DNA copy number and methylation in adolescent rats
Genki Mizuno, Hiroya Yamada, Eiji Munetsuna, et al.
Nutrition Research (2022) Vol. 110, pp. 57-65
Closed Access | Times Cited: 2

Association between the Extent of Peripheral Blood DNA Methylation of HIF3A and Accumulation of Adiposity in community-dwelling Women: The Yakumo Study
Genki Mizuno, Hiroya Yamada, Eiji Munetsuna, et al.
Endocrine Research (2022) Vol. 47, Iss. 3-4, pp. 130-137
Closed Access | Times Cited: 1

Interaction between Prenatal and Postnatal Exposure to High-Fructose Corn Syrup Increases Gene Expression of <i>Tnfa</i> in Hippocampus of Offspring
Mirai Yamazaki, Hiroya Yamada, Eiji Munetsuna, et al.
Journal of Nutritional Science and Vitaminology (2023) Vol. 69, Iss. 4, pp. 237-242
Open Access

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