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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Down syndrome is an oxidative phosphorylation disorder
M. Pilar Bayona‐Bafaluy, Nuria Garrido-Pérez, Patricia Meade, et al.
Redox Biology (2021) Vol. 41, pp. 101871-101871
Open Access | Times Cited: 24

Showing 24 citing articles:

Increased dosage of DYRK1A leads to congenital heart defects in a mouse model of Down syndrome
Eva Lana‐Elola, Rifdat Aoidi, Miriam Llorian, et al.
Science Translational Medicine (2024) Vol. 16, Iss. 731
Open Access | Times Cited: 19
Mitochondria Can Cross Cell Boundaries: An Overview of the Biological Relevance, Pathophysiological Implications and Therapeutic Perspectives of Intercellular Mitochondrial Transfer
Daniela Valenti, Rosa Anna Vacca, Loredana Moro, et al.
International Journal of Molecular Sciences (2021) Vol. 22, Iss. 15, pp. 8312-8312
Open Access | Times Cited: 82
Complex II ambiguities—FADH2 in the electron transfer system
Erich Gnaiger
Journal of Biological Chemistry (2023) Vol. 300, Iss. 1, pp. 105470-105470
Open Access | Times Cited: 23
Mitochondrial Dysfunction in Down Syndrome: From Pathology to Therapy
Kai-Leng Tan, Han-Chung Lee, Pike-See Cheah, et al.
Neuroscience (2022) Vol. 511, pp. 1-12
Closed Access | Times Cited: 22
Brain Mitochondrial Bioenergetics in Genetic Neurodevelopmental Disorders: Focus on Down, Rett and Fragile X Syndromes
Daniela Valenti, Rosa Anna Vacca
International Journal of Molecular Sciences (2023) Vol. 24, Iss. 15, pp. 12488-12488
Open Access | Times Cited: 12
Profiling hippocampal neuronal populations reveals unique gene expression mosaics reflective of connectivity-based degeneration in the Ts65Dn mouse model of Down syndrome and Alzheimer’s disease
Melissa J. Alldred, Kyrillos W. Ibrahim, Harshitha Pidikiti, et al.
Frontiers in Molecular Neuroscience (2025) Vol. 18
Open Access
Role of cystathionine-β-synthase and hydrogen sulfide in down syndrome
Csaba Szabó
Neurotherapeutics (2025), pp. e00584-e00584
Open Access
Mitochondrial Imbalance in Down Syndrome: A Driver of Accelerated Brain Aging?
Xinxin Zuo
Aging and Disease (2025)
Open Access
Oxidative Phosphorylation Is Dysregulated Within the Basocortical Circuit in a 6-month old Mouse Model of Down Syndrome and Alzheimer’s Disease
Melissa J. Alldred, Sang H. Lee, Grace E. Stutzmann, et al.
Frontiers in Aging Neuroscience (2021) Vol. 13
Open Access | Times Cited: 21
Impaired Brain Mitochondrial Bioenergetics in the Ts65Dn Mouse Model of Down Syndrome Is Restored by Neonatal Treatment with the Polyphenol 7,8-Dihydroxyflavone
Daniela Valenti, Fiorenza Stagni, Marco Emili, et al.
Antioxidants (2021) Vol. 11, Iss. 1, pp. 62-62
Open Access | Times Cited: 16
Therapeutics for mitochondrial dysfunction-linked diseases in Down syndrome
Bani Bandana Ganguly, Nitin N. Kadam
Mitochondrion (2022) Vol. 68, pp. 25-43
Open Access | Times Cited: 12
Transition from Animal-Based to Human Induced Pluripotent Stem Cells (iPSCs)-Based Models of Neurodevelopmental Disorders: Opportunities and Challenges
Sara Guerreiro, Patrı́cia Maciel
Cells (2023) Vol. 12, Iss. 4, pp. 538-538
Open Access | Times Cited: 6
Evidence of Energy Metabolism Alterations in Cultured Neonatal Astrocytes Derived from the Ts65Dn Mouse Model of Down Syndrome
Bruna Lancia Zampieri, Alberto C. S. Costa
Brain Sciences (2022) Vol. 12, Iss. 1, pp. 83-83
Open Access | Times Cited: 8
Fatty Acids: A Safe Tool for Improving Neurodevelopmental Alterations in Down Syndrome?
Carmen Martı́nez-Cué, Renata Bartesaghi
Nutrients (2022) Vol. 14, Iss. 14, pp. 2880-2880
Open Access | Times Cited: 8
Oxidative-Stress-Associated Proteostasis Disturbances and Increased DNA Damage in the Hippocampal Granule Cells of the Ts65Dn Model of Down Syndrome
Alba Puente-Bedia, Marı́a T. Berciano, Carmen Martı́nez-Cué, et al.
Antioxidants (2022) Vol. 11, Iss. 12, pp. 2438-2438
Open Access | Times Cited: 8
Single‐cell landscape analysis reveals systematic senescence in mammalian Down syndrome
Yao Chen, Yanyu Xiao, Yanye Zhang, et al.
Clinical and Translational Medicine (2023) Vol. 13, Iss. 7
Open Access | Times Cited: 3
The many “Neurofaces” of Prohibitins 1 and 2: Crucial for the healthy brain, dysregulated in numerous brain disorders
Hans‐Gert Bernstein, Karl‐Heinz Smalla, Gerburg Keilhoff, et al.
Journal of Chemical Neuroanatomy (2023) Vol. 132, pp. 102321-102321
Closed Access | Times Cited: 3
A human fetal cerebellar map of the late second trimester reveals developmental molecular characteristics and abnormality in trisomy 21
Hongmin Yu, Yun Liu, Fanqing Xu, et al.
Cell Reports (2024) Vol. 43, Iss. 8, pp. 114586-114586
Open Access
Integrated analysis of immunometabolic interactions in Down syndrome
Lucas A. Gillenwater, Matthew D. Galbraith, Angela L. Rachubinski, et al.
Science Advances (2024) Vol. 10, Iss. 50
Open Access
Single-Nucleus Profiling Identifies Accelerated Oligodendrocyte Precursor Cell Senescence in a Mouse Model of Down Syndrome
Bianca Rusu, Bharti Kukreja, Taiyi Wu, et al.
eNeuro (2023) Vol. 10, Iss. 8, pp. ENEURO.0147-23.2023
Open Access | Times Cited: 1
Genetic and Molecular Tools for the Clinical Diagnosis of Down Syndrome
Mónica Paulina Manzano Vela, Dennis Renato Manzano Vela, Ana Carola Flores Mancheno, et al.
Salud Ciencia y Tecnología (2024) Vol. 5, pp. 1027-1027
Closed Access
Disarrayed mitochondrial function on pathobiology in Down syndrome and targeted therapeutics
Bani Bandana Ganguly
Elsevier eBooks (2022), pp. 219-243
Closed Access | Times Cited: 1
Congenital heart defects in Down syndrome are caused by increased dosage of DYRK1A
Eva Lana‐Elola, Rifdat Aoidi, Miriam Llorian, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2023)
Open Access
Senescent hearts from male Ts65Dn mice exhibit preserved function but altered size and nicotinamide adenine dinucleotide pathway signaling
Josef Brandauer, Candace N. Receno, Cynthia Anyaoku, et al.
AJP Regulatory Integrative and Comparative Physiology (2023) Vol. 326, Iss. 2, pp. R176-R183
Closed Access
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