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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

N6-methyladenosine of Spi2a attenuates inflammation and sepsis-associated myocardial dysfunction in mice
Xiangyu Wang, Yan Ding, Ran Li, et al.
Nature Communications (2023) Vol. 14, Iss. 1
Open Access | Times Cited: 34

Showing 1-25 of 34 citing articles:

S100A8/A9hi neutrophils induce mitochondrial dysfunction and PANoptosis in endothelial cells via mitochondrial complex I deficiency during sepsis
Yanghanzhao Wang, Yuxin Shi, Yuwen Shao, et al.
Cell Death and Disease (2024) Vol. 15, Iss. 6
Open Access | Times Cited: 17
METTL14 regulates microglia/macrophage polarization and NLRP3 inflammasome activation after ischemic stroke by the KAT3B-STING axis
Yamei Li, Jiacen Li, Qian Yu, et al.
Neurobiology of Disease (2023) Vol. 185, pp. 106253-106253
Open Access | Times Cited: 33
METTL14 contributes to acute lung injury by stabilizing NLRP3 expression in an IGF2BP2-dependent manner
Fei Cao, Guojun Chen, Yixin Xu, et al.
Cell Death and Disease (2024) Vol. 15, Iss. 1
Open Access | Times Cited: 14
m6A epitranscriptomic and epigenetic crosstalk in cardiac fibrosis
Zhiyan Liu, Li-Chan Lin, Zhenyu Liu, et al.
Molecular Therapy (2024) Vol. 32, Iss. 4, pp. 878-889
Closed Access | Times Cited: 12
Pulsatile Flow Increases METTL14-induced m6A modification and attenuates septic cardiomyopathy: an experimental study
Shenyu Zhu, Kai Wang, Zhexuan Yu, et al.
International Journal of Surgery (2024)
Open Access | Times Cited: 6
RNA m6A methylation regulators in sepsis
Lin Zhu, Hairong Zhang, Xiaoyu Zhang, et al.
Molecular and Cellular Biochemistry (2023) Vol. 479, Iss. 9, pp. 2165-2180
Closed Access | Times Cited: 16
Sepsis induced cardiotoxicity by promoting cardiomyocyte cuproptosis
Jingru Yan, Zhangyi Li, Yilan Li, et al.
Biochemical and Biophysical Research Communications (2023) Vol. 690, pp. 149245-149245
Closed Access | Times Cited: 13
Fullerenol as a novel therapeutic agent for sepsis-induced cardiomyocytes damage
Tingjun Zhang, Ling Chen, Xuefeng Ding, et al.
Applied Physics A (2024) Vol. 130, Iss. 3
Open Access | Times Cited: 4
Clinical Perspectives in Epitranscriptomics
Eloy Santos-Pujol, Carlos Quero-Dotor, Manel Esteller
Current Opinion in Genetics & Development (2024) Vol. 87, pp. 102209-102209
Open Access | Times Cited: 4
eIF3a function in immunity and protection against severe sepsis by regulating B cell quantity and function through m6A modification
Qianying Ouyang, Jiajia Cui, Yang Wang, et al.
Acta Pharmaceutica Sinica B (2025)
Open Access
The m6A modification of SOX18 leads to increased PTX3 and cardiomyocyte pyroptosis in sepsis-induced cardiomyopathy
He Sun, Xinan Qiao, Xiangyan Peng, et al.
Theranostics (2025) Vol. 15, Iss. 8, pp. 3532-3550
Open Access
Microfluidic magnetic droplet-based chemiluminescence enzyme immunoassay for multiplex sepsis biomarker screening
Jiayue Huang, Jinbo Li, Nan Wu, et al.
The Analyst (2025)
Closed Access
Enantiomer-Dependent Uptake of Chiral Nanoparticles in Macrophages Modulates the Inflammatory Response through the NF-κB Pathway
Jinghua Hao, Yijin Tian, Jie Tang, et al.
Environmental Science & Technology (2025)
Closed Access
Extremely low-frequency electromagnetic field induces acetylation of heat shock proteins and enhances protein folding
Zhizhou Huang, Mikako Ito, Shaochuan Zhang, et al.
Ecotoxicology and Environmental Safety (2023) Vol. 264, pp. 115482-115482
Open Access | Times Cited: 10
PARK7 Is Required to Protect Epithelia Against Damage in Oral Lichen Planus
Yufeng Fan, Yiran Hao, Yan Ding, et al.
Oral Diseases (2025)
Closed Access
N6-methyladenosine modification: Regulatory mechanisms and therapeutic potential in sepsis
Wei Wang, Huaili Wang, Tongwen Sun
Biomedicine & Pharmacotherapy (2023) Vol. 168, pp. 115719-115719
Open Access | Times Cited: 8
Impacts of epigenetic reprogramming on innate immunity
Jie Fu, Yizhen Wang
Deleted Journal (2024) Vol. 1
Open Access | Times Cited: 2
The potential of RNA methylation in the treatment of cardiovascular diseases
Kai Wang, Yuqin Wang, Yinghui Li, et al.
iScience (2024) Vol. 27, Iss. 8, pp. 110524-110524
Open Access | Times Cited: 2
Cytosolic serpins act in a cytoprotective feedback loop that limits ESX-1-dependent death of Mycobacterium marinum -infected macrophages
Esther Nobs, Katie Laschanzky, Kristina Munke, et al.
mBio (2024) Vol. 15, Iss. 9
Open Access | Times Cited: 2
Post-translational modifications in sepsis-induced organ dysfunction: mechanisms and implications
Lin Song, Wei Jiang, Lin Hua, et al.
Frontiers in Immunology (2024) Vol. 15
Open Access | Times Cited: 2
Role of N6‐methyladenosine RNA modification in cancer
Yi Qu, Nannan Gao, Shengwei Zhang, et al.
MedComm (2024) Vol. 5, Iss. 9
Open Access | Times Cited: 2
Obesity Enables NLRP3 Activation and Induces Myocardial Fibrosis via Hyperacetylation of HADHa
Yan Deng, Xin Liu, Min Xie, et al.
Diabetes (2023) Vol. 72, Iss. 11, pp. 1597-1608
Open Access | Times Cited: 6
N6-Methyladenosine (m6A) Modification in Natural Immune Cell-Mediated Inflammatory Diseases
Yan Teng, Yi Jin, Junnian Chen, et al.
Journal of Innate Immunity (2023) Vol. 15, Iss. 1, pp. 804-821
Open Access | Times Cited: 6
Circulating Exosomes from Septic Mice Activate NF-κB/MIR17HG Pathway in Macrophages
Shao‐Chun Wu, Cheng‐Shyuan Rau, Yi-Chan Wu, et al.
Biomedicines (2024) Vol. 12, Iss. 3, pp. 534-534
Open Access | Times Cited: 1
m6A epitranscriptomic modification of inflammation in cardiovascular disease
Yun-Sen Zhang, Zhiyan Liu, Zhenyu Liu, et al.
International Immunopharmacology (2024) Vol. 134, pp. 112222-112222
Closed Access | Times Cited: 1
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