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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Expanded GGGGCC repeat RNA associated with amyotrophic lateral sclerosis and frontotemporal dementia causes neurodegeneration
Zihui Xu, Mickaël Poidevin, Xuekun Li, et al.
Proceedings of the National Academy of Sciences (2013) Vol. 110, Iss. 19, pp. 7778-7783
Open Access | Times Cited: 335

Showing 1-25 of 335 citing articles:

Decoding ALS: from genes to mechanism
J. Paul Taylor, Robert H. Brown, Don W. Cleveland
Nature (2016) Vol. 539, Iss. 7628, pp. 197-206
Open Access | Times Cited: 1821
Converging Mechanisms in ALS and FTD: Disrupted RNA and Protein Homeostasis
Shuo‐Chien Ling, Magdalini Polymenidou, Don W. Cleveland
Neuron (2013) Vol. 79, Iss. 3, pp. 416-438
Open Access | Times Cited: 1607
The C9orf72 repeat expansion disrupts nucleocytoplasmic transport
Ke Zhang, Christopher J. Donnelly, Aaron R. Haeusler, et al.
Nature (2015) Vol. 525, Iss. 7567, pp. 56-61
Open Access | Times Cited: 922
C9orf72 nucleotide repeat structures initiate molecular cascades of disease
Aaron R. Haeusler, Christopher J. Donnelly, Goran Periz, et al.
Nature (2014) Vol. 507, Iss. 7491, pp. 195-200
Open Access | Times Cited: 921
RNA Toxicity from the ALS/FTD C9ORF72 Expansion Is Mitigated by Antisense Intervention
Christopher J. Donnelly, Ping‐Wu Zhang, Jacqueline T. Pham, et al.
Neuron (2013) Vol. 80, Iss. 2, pp. 415-428
Open Access | Times Cited: 851
GGGGCC repeat expansion in C9orf72 compromises nucleocytoplasmic transport
Brian D. Freibaum, Yubing Lu, Rodrigo López‐González, et al.
Nature (2015) Vol. 525, Iss. 7567, pp. 129-133
Open Access | Times Cited: 791
RAN proteins and RNA foci from antisense transcripts in C9ORF72 ALS and frontotemporal dementia
Tao Zu, Yuanjing Liu, Mónica Báñez-Coronel, et al.
Proceedings of the National Academy of Sciences (2013) Vol. 110, Iss. 51
Open Access | Times Cited: 743
C9orf72 repeat expansions cause neurodegeneration in Drosophila through arginine-rich proteins
Sarah Mizielinska, Sebastian Grönke, Teresa Niccoli, et al.
Science (2014) Vol. 345, Iss. 6201, pp. 1192-1194
Open Access | Times Cited: 695
C9orf72 Dipeptide Repeats Impair the Assembly, Dynamics, and Function of Membrane-Less Organelles
Kyung‐Ha Lee, Peipei Zhang, Hong Joo Kim, et al.
Cell (2016) Vol. 167, Iss. 3, pp. 774-788.e17
Open Access | Times Cited: 670
Targeting RNA Foci in iPSC-Derived Motor Neurons from ALS Patients with a C9ORF72 Repeat Expansion
Dhruv Sareen, Jacqueline G. O’Rourke, Pratap Meera, et al.
Science Translational Medicine (2013) Vol. 5, Iss. 208
Open Access | Times Cited: 634
C9orf72-mediated ALS and FTD: multiple pathways to disease
Rubika Balendra, Adrian M. Isaacs
Nature Reviews Neurology (2018) Vol. 14, Iss. 9, pp. 544-558
Open Access | Times Cited: 616
Antisense transcripts of the expanded C9ORF72 hexanucleotide repeat form nuclear RNA foci and undergo repeat-associated non-ATG translation in c9FTD/ALS
Tania F. Gendron, Kevin F. Bieniek, Yong‐Jie Zhang, et al.
Acta Neuropathologica (2013) Vol. 126, Iss. 6, pp. 829-844
Open Access | Times Cited: 559
Targeted degradation of sense and antisense C9orf72 RNA foci as therapy for ALS and frontotemporal degeneration
Clotilde Lagier‐Tourenne, Michael W. Baughn, Frank Rigo, et al.
Proceedings of the National Academy of Sciences (2013) Vol. 110, Iss. 47
Open Access | Times Cited: 534
Antisense Proline-Arginine RAN Dipeptides Linked to C9ORF72-ALS/FTD Form Toxic Nuclear Aggregates that Initiate In Vitro and In Vivo Neuronal Death
Xinmei Wen, Wenzhi Tan, Thomas Westergard, et al.
Neuron (2014) Vol. 84, Iss. 6, pp. 1213-1225
Open Access | Times Cited: 521
Gain of Toxicity from ALS/FTD-Linked Repeat Expansions in C9ORF72 Is Alleviated by Antisense Oligonucleotides Targeting GGGGCC-Containing RNAs
Jie Jiang, Qiang Zhu, Tania F. Gendron, et al.
Neuron (2016) Vol. 90, Iss. 3, pp. 535-550
Open Access | Times Cited: 502
Hexanucleotide Repeats in ALS/FTD Form Length-Dependent RNA Foci, Sequester RNA Binding Proteins, and Are Neurotoxic
Youn‐Bok Lee, Han-Jou Chen, João Peres, et al.
Cell Reports (2013) Vol. 5, Iss. 5, pp. 1178-1186
Open Access | Times Cited: 466
Stress Granule Assembly Disrupts Nucleocytoplasmic Transport
Ke Zhang, J. Gavin Daigle, Kathleen M. Cunningham, et al.
Cell (2018) Vol. 173, Iss. 4, pp. 958-971.e17
Open Access | Times Cited: 384
C9ORF72 repeat expansions in mice cause TDP-43 pathology, neuronal loss, and behavioral deficits
Jeannie Chew, Tania F. Gendron, Mercedes Prudencio, et al.
Science (2015) Vol. 348, Iss. 6239, pp. 1151-1154
Open Access | Times Cited: 371
Direct conversion of patient fibroblasts demonstrates non-cell autonomous toxicity of astrocytes to motor neurons in familial and sporadic ALS
Kathrin Meyer, Laura Ferraiuolo, Carlos J. Miranda, et al.
Proceedings of the National Academy of Sciences (2013) Vol. 111, Iss. 2, pp. 829-832
Open Access | Times Cited: 324
C9orf72 FTLD/ALS-associated Gly-Ala dipeptide repeat proteins cause neuronal toxicity and Unc119 sequestration
Stephanie May, Daniel Hornburg, Martin H. Schludi, et al.
Acta Neuropathologica (2014) Vol. 128, Iss. 4, pp. 485-503
Open Access | Times Cited: 310
Drosophila as an In Vivo Model for Human Neurodegenerative Disease
Leeanne McGurk, Amit Berson, Nancy M. Bonini
Genetics (2015) Vol. 201, Iss. 2, pp. 377-402
Open Access | Times Cited: 302
ALS Genetics: Gains, Losses, and Implications for Future Therapies
Garam Kım, Olivia Gautier, Eduardo Tassoni-Tsuchida, et al.
Neuron (2020) Vol. 108, Iss. 5, pp. 822-842
Open Access | Times Cited: 301
Sequestration of multiple RNA recognition motif-containing proteins by C9orf72 repeat expansions
Johnathan Cooper‐Knock, Matthew J. Walsh, Adrian Higginbottom, et al.
Brain (2014) Vol. 137, Iss. 7, pp. 2040-2051
Open Access | Times Cited: 288
C9orf72 BAC Transgenic Mice Display Typical Pathologic Features of ALS/FTD
Jacqueline G. O’Rourke, Laurent Bogdanik, A.K.M. Ghulam Muhammad, et al.
Neuron (2015) Vol. 88, Iss. 5, pp. 892-901
Open Access | Times Cited: 284
The C9ORF72 GGGGCC expansion forms RNA G-quadruplex inclusions and sequesters hnRNP H to disrupt splicing in ALS brains
Erin G. Conlon, Lei Lü, Aarti Sharma, et al.
eLife (2016) Vol. 5
Open Access | Times Cited: 263
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