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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

RAN proteins and RNA foci from antisense transcripts in C9ORF72 ALS and frontotemporal dementia
Tao Zu, Yuanjing Liu, Mónica Báñez-Coronel, et al.
Proceedings of the National Academy of Sciences (2013) Vol. 110, Iss. 51
Open Access | Times Cited: 743

Showing 1-25 of 743 citing articles:

Protein Phase Separation: A New Phase in Cell Biology
Steven Boeynaems, Simon Alberti, Nicolas L. Fawzi, et al.
Trends in Cell Biology (2018) Vol. 28, Iss. 6, pp. 420-435
Open Access | Times Cited: 1831
Decoding ALS: from genes to mechanism
J. Paul Taylor, Robert H. Brown, Don W. Cleveland
Nature (2016) Vol. 539, Iss. 7628, pp. 197-206
Open Access | Times Cited: 1820
C9orf72 nucleotide repeat structures initiate molecular cascades of disease
Aaron R. Haeusler, Christopher J. Donnelly, Goran Periz, et al.
Nature (2014) Vol. 507, Iss. 7491, pp. 195-200
Open Access | Times Cited: 921
C9orf72 repeat expansions cause neurodegeneration in Drosophila through arginine-rich proteins
Sarah Mizielinska, Sebastian Grönke, Teresa Niccoli, et al.
Science (2014) Vol. 345, Iss. 6201, pp. 1192-1194
Open Access | Times Cited: 695
C9orf72 Dipeptide Repeats Impair the Assembly, Dynamics, and Function of Membrane-Less Organelles
Kyung‐Ha Lee, Peipei Zhang, Hong Joo Kim, et al.
Cell (2016) Vol. 167, Iss. 3, pp. 774-788.e17
Open Access | Times Cited: 670
C9orf72-mediated ALS and FTD: multiple pathways to disease
Rubika Balendra, Adrian M. Isaacs
Nature Reviews Neurology (2018) Vol. 14, Iss. 9, pp. 544-558
Open Access | Times Cited: 615
Poly-dipeptides encoded by the C9orf72 repeats bind nucleoli, impede RNA biogenesis, and kill cells
Ilmin Kwon, Siheng Xiang, Masato Kato, et al.
Science (2014) Vol. 345, Iss. 6201, pp. 1139-1145
Open Access | Times Cited: 613
Modifiers of C9orf72 dipeptide repeat toxicity connect nucleocytoplasmic transport defects to FTD/ALS
Ana Jovičić, Jérôme Mertens, Steven Boeynaems, et al.
Nature Neuroscience (2015) Vol. 18, Iss. 9, pp. 1226-1229
Open Access | Times Cited: 593
Antisense Proline-Arginine RAN Dipeptides Linked to C9ORF72-ALS/FTD Form Toxic Nuclear Aggregates that Initiate In Vitro and In Vivo Neuronal Death
Xinmei Wen, Wenzhi Tan, Thomas Westergard, et al.
Neuron (2014) Vol. 84, Iss. 6, pp. 1213-1225
Open Access | Times Cited: 521
Gain of Toxicity from ALS/FTD-Linked Repeat Expansions in C9ORF72 Is Alleviated by Antisense Oligonucleotides Targeting GGGGCC-Containing RNAs
Jie Jiang, Qiang Zhu, Tania F. Gendron, et al.
Neuron (2016) Vol. 90, Iss. 3, pp. 535-550
Open Access | Times Cited: 502
RNA self-assembly contributes to stress granule formation and defining the stress granule transcriptome
Briana Van Treeck, David S.W. Protter, Tyler Matheny, et al.
Proceedings of the National Academy of Sciences (2018) Vol. 115, Iss. 11, pp. 2734-2739
Open Access | Times Cited: 474
Spontaneous driving forces give rise to protein−RNA condensates with coexisting phases and complex material properties
Steven Boeynaems, Alex S. Holehouse, Venera Weinhardt, et al.
Proceedings of the National Academy of Sciences (2019) Vol. 116, Iss. 16, pp. 7889-7898
Open Access | Times Cited: 469
Toxic PR Poly-Dipeptides Encoded by the C9orf72 Repeat Expansion Target LC Domain Polymers
Yi Lin, Eiichiro Mori, Masato Kato, et al.
Cell (2016) Vol. 167, Iss. 3, pp. 789-802.e12
Open Access | Times Cited: 431
Poly(GR) in C9ORF72 -Related ALS/FTD Compromises Mitochondrial Function and Increases Oxidative Stress and DNA Damage in iPSC-Derived Motor Neurons
Rodrigo López‐González, Yubing Lu, Tania F. Gendron, et al.
Neuron (2016) Vol. 92, Iss. 2, pp. 383-391
Open Access | Times Cited: 384
Tandem repeats mediating genetic plasticity in health and disease
Anthony J. Hannan
Nature Reviews Genetics (2018) Vol. 19, Iss. 5, pp. 286-298
Closed Access | Times Cited: 382
In Situ Structure of Neuronal C9orf72 Poly-GA Aggregates Reveals Proteasome Recruitment
Qiang Guo, Carina Lehmer, Antonio Martínez-Sánchez, et al.
Cell (2018) Vol. 172, Iss. 4, pp. 696-705.e12
Open Access | Times Cited: 379
Loss of C9 ORF 72 impairs autophagy and synergizes with polyQ Ataxin‐2 to induce motor neuron dysfunction and cell death
Chantal Sellier, Maria‐Letizia Campanari, Camille Corbier, et al.
The EMBO Journal (2016) Vol. 35, Iss. 12, pp. 1276-1297
Open Access | Times Cited: 366
The C9orf72 protein interacts with Rab1a and the ULK1 complex to regulate initiation of autophagy
Christopher P Webster, Emma F. Smith, Claudia S. Bauer, et al.
The EMBO Journal (2016) Vol. 35, Iss. 15, pp. 1656-1676
Open Access | Times Cited: 362
Non-AUG translation: a new start for protein synthesis in eukaryotes
Michael G. Kearse, Jeremy E. Wilusz
Genes & Development (2017) Vol. 31, Iss. 17, pp. 1717-1731
Open Access | Times Cited: 361
The frontotemporal dementia-motor neuron disease continuum
James R. Burrell, Glenda M. Halliday, Jillian J. Kril, et al.
The Lancet (2016) Vol. 388, Iss. 10047, pp. 919-931
Closed Access | Times Cited: 344
C9orf72 BAC Mouse Model with Motor Deficits and Neurodegenerative Features of ALS/FTD
Yuanjing Liu, Amrutha Pattamatta, Tao Zu, et al.
Neuron (2016) Vol. 90, Iss. 3, pp. 521-534
Open Access | Times Cited: 344
Discovery of a Biomarker and Lead Small Molecules to Target r(GGGGCC)-Associated Defects in c9FTD/ALS
Zhaoming Su, Yong‐Jie Zhang, Tania F. Gendron, et al.
Neuron (2014) Vol. 83, Iss. 5, pp. 1043-1050
Open Access | Times Cited: 341
RAN Translation in Huntington Disease
Mónica Báñez-Coronel, Fatma Ayhan, Alex D. Tarabochia, et al.
Neuron (2015) Vol. 88, Iss. 4, pp. 667-677
Open Access | Times Cited: 320
Aggregation-prone c9FTD/ALS poly(GA) RAN-translated proteins cause neurotoxicity by inducing ER stress
Yong‐Jie Zhang, Karen Jansen‐West, Ya-Fei Xu, et al.
Acta Neuropathologica (2014) Vol. 128, Iss. 4, pp. 505-524
Open Access | Times Cited: 319
Molecular neuropathology of frontotemporal dementia: insights into disease mechanisms from postmortem studies
Ian R. Mackenzie, Manuela Neumann
Journal of Neurochemistry (2016) Vol. 138, Iss. S1, pp. 54-70
Open Access | Times Cited: 315
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