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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Complexity in genetic cardiomyopathies and new approaches for mechanism-based precision medicine
Michael J. Greenberg, Jil C. Tardiff
The Journal of General Physiology (2021) Vol. 153, Iss. 3
Open Access | Times Cited: 36

Showing 1-25 of 36 citing articles:

Drug specificity and affinity are encoded in the probability of cryptic pocket opening in myosin motor domains
Artur Meller, Jeffrey M. Lotthammer, Louis G. Smith, et al.
eLife (2023) Vol. 12
Open Access | Times Cited: 28
Cardiac myosin contraction and mechanotransduction in health and disease
Samantha K. Barrick, Michael J. Greenberg
Journal of Biological Chemistry (2021) Vol. 297, Iss. 5, pp. 101297-101297
Open Access | Times Cited: 54
Translation of New and Emerging Therapies for Genetic Cardiomyopathies
Adam Helms, Andrea D. Thompson, Sharlene M. Day
JACC Basic to Translational Science (2021) Vol. 7, Iss. 1, pp. 70-83
Open Access | Times Cited: 45
Mechanisms of myosin II force generation. Insights from novel experimental techniques and approaches
Dilson E. Rassier, Alf Månsson
Physiological Reviews (2024) Vol. 105, Iss. 1, pp. 1-93
Closed Access | Times Cited: 5
Sex-specific response to A1BG loss results in female dilated cardiomyopathy
James I. Emerson, Wei Shi, Frank L. Conlon
Biology of Sex Differences (2025) Vol. 16, Iss. 1
Open Access
Unraveling the Complexities of Biventricular Hypertrophic Cardiomyopathy: A Case of Right Ventricular Outflow Tract Obstruction Illuminated by Cardiac Magnetic Resonance Imaging
Digvijay Nalawade, S.K. Malani, Chigullapalli Sridevi, et al.
Journal of the Practice of Cardiovascular Sciences (2025) Vol. 11, Iss. 1, pp. 83-85
Open Access
Mechanical dysfunction of the sarcomere induced by a pathogenic mutation in troponin T drives cellular adaptation
Sarah R. Clippinger, Paige E. Cloonan, Wei Wang, et al.
The Journal of General Physiology (2021) Vol. 153, Iss. 5
Open Access | Times Cited: 21
Critical Evaluation of Current Hypotheses for the Pathogenesis of Hypertrophic Cardiomyopathy
Marko Ušaj, Luisa Moretto, Alf Månsson
International Journal of Molecular Sciences (2022) Vol. 23, Iss. 4, pp. 2195-2195
Open Access | Times Cited: 15
Dilated cardiomyopathy–associated skeletal muscle actin (ACTA1) mutation R256H disrupts actin structure and function and causes cardiomyocyte hypocontractility
Ankit Garg, Silvia Jansen, Lina Greenberg, et al.
Proceedings of the National Academy of Sciences (2024) Vol. 121, Iss. 46
Open Access | Times Cited: 3
Biomechanical signals regulating the structure of the heart
Joseph D. Powers, Andrew D. McCulloch
Current Opinion in Physiology (2022) Vol. 25, pp. 100482-100482
Open Access | Times Cited: 12
Dilated cardiomyopathy-associated skeletal muscle actin (ACTA1) mutation R256H disrupts actin structure and function and causes cardiomyocyte hypocontractility
Ankit Garg, Silvia Jansen, Rui Zhang, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access | Times Cited: 2
Harnessing molecular mechanism for precision medicine in dilated cardiomyopathy caused by a mutation in troponin T
Lina Greenberg, W. Tom Stump, Zongtao Lin, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access | Times Cited: 2
Myosin-Catalyzed ATP Hydrolysis in the Presence of Disease-Causing Mutations: Mavacamten as a Way to Repair Mechanism
Ananya Chakraborti, Jil C. Tardiff, Steven D. Schwartz
The Journal of Physical Chemistry B (2024) Vol. 128, Iss. 19, pp. 4716-4727
Closed Access | Times Cited: 2
Excitation–contraction coupling in cardiac, skeletal, and smooth muscle
Robert T. Dirksen, David Eisner, Eduardo Rı́os, et al.
The Journal of General Physiology (2022) Vol. 154, Iss. 9
Open Access | Times Cited: 10
A troponin T variant linked with pediatric dilated cardiomyopathy reduces the coupling of thin filament activation to myosin and calcium binding
Samantha K. Barrick, Lina Greenberg, Michael J. Greenberg
Molecular Biology of the Cell (2021) Vol. 32, Iss. 18, pp. 1677-1689
Open Access | Times Cited: 13
Functional assays reveal the pathogenic mechanism of a de novo tropomyosin variant identified in patient with dilated cardiomyopathy
Samantha K. Barrick, Ankit Garg, Lina Greenberg, et al.
Journal of Molecular and Cellular Cardiology (2023) Vol. 176, pp. 58-67
Open Access | Times Cited: 5
Advances in Hypertrophic Cardiomyopathy Disease Modelling Using hiPSC-Derived Cardiomyocytes
Saif Dababneh, Homa Hamledari, Yasaman Maaref, et al.
Canadian Journal of Cardiology (2023) Vol. 40, Iss. 5, pp. 766-776
Closed Access | Times Cited: 5
Advances in Multi-Modality Imaging in Hypertrophic Cardiomyopathy
Fraser C. Goldie, Matthew M.Y. Lee, Caroline Coats, et al.
Journal of Clinical Medicine (2024) Vol. 13, Iss. 3, pp. 842-842
Open Access | Times Cited: 1
Computational and biophysical determination of pathogenicity of variants of unknown significance in cardiac thin filament
Allison B. Mason, Melissa L. Lynn, Anthony Baldo, et al.
JCI Insight (2021) Vol. 6, Iss. 23
Open Access | Times Cited: 9
Insights into the Mechanism of the Cardiac Drug Omecamtiv Mecarbil─A Computational Study
Ananya Chakraborti, Jil C. Tardiff, Steven D. Schwartz
The Journal of Physical Chemistry B (2022) Vol. 126, Iss. 48, pp. 10069-10082
Open Access | Times Cited: 6
Assessing Cardiac Contractility From Single Molecules to Whole Hearts
Ankit Garg, Kory J. Lavine, Michael J. Greenberg
JACC Basic to Translational Science (2023) Vol. 9, Iss. 3, pp. 414-439
Open Access | Times Cited: 3
The effect of Mg2+ on Ca2+ binding to cardiac troponin C in hypertrophic cardiomyopathy associated TNNC1 variants
Kaveh Rayani, Eric R. Hantz, Omid Haji‐Ghassemi, et al.
FEBS Journal (2022) Vol. 289, Iss. 23, pp. 7446-7465
Open Access | Times Cited: 5
Single-molecule mechanics and kinetics of cardiac myosin interacting with regulated thin filaments
Sarah R. Clippinger Schulte, Brent Scott, Samantha K. Barrick, et al.
Biophysical Journal (2023) Vol. 122, Iss. 12, pp. 2544-2555
Open Access | Times Cited: 2
The prevalence of mutations in the MYBPC3 gene in Russian patients with hypertrophic cardiomyopathy
Anna L. Klass, Н. С. Крылова, A.V. Lysenko, et al.
Molecular Genetics Microbiology and Virology (Russian) (2023) Vol. 41, Iss. 1, pp. 18-18
Closed Access | Times Cited: 2
Structural dynamics of the intrinsically disordered linker region of cardiac troponin T
Jasmine Cubuk, Lina Greenberg, Akiva E. Greenberg, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access
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