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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Nucleolar stress and impaired stress granule formation contribute to C9orf72 RAN translation-induced cytotoxicity
Zhouteng Tao, Hongfeng Wang, Xia Qin, et al.
Human Molecular Genetics (2015) Vol. 24, Iss. 9, pp. 2426-2441
Open Access | Times Cited: 223

Showing 1-25 of 223 citing articles:

C9orf72-mediated ALS and FTD: multiple pathways to disease
Rubika Balendra, Adrian M. Isaacs
Nature Reviews Neurology (2018) Vol. 14, Iss. 9, pp. 544-558
Open Access | Times Cited: 616
Phase Separation of C9orf72 Dipeptide Repeats Perturbs Stress Granule Dynamics
Steven Boeynaems, Elke Bogaert, Dénes Kovács, et al.
Molecular Cell (2017) Vol. 65, Iss. 6, pp. 1044-1055.e5
Open Access | Times Cited: 507
Loss of C9 ORF 72 impairs autophagy and synergizes with polyQ Ataxin‐2 to induce motor neuron dysfunction and cell death
Chantal Sellier, Maria‐Letizia Campanari, Camille Corbier, et al.
The EMBO Journal (2016) Vol. 35, Iss. 12, pp. 1276-1297
Open Access | Times Cited: 366
Distinct brain transcriptome profiles in C9orf72-associated and sporadic ALS
Mercedes Prudencio, Véronique Belzil, Ranjan Batra, et al.
Nature Neuroscience (2015) Vol. 18, Iss. 8, pp. 1175-1182
Open Access | Times Cited: 363
ALS Genetics: Gains, Losses, and Implications for Future Therapies
Garam Kım, Olivia Gautier, Eduardo Tassoni-Tsuchida, et al.
Neuron (2020) Vol. 108, Iss. 5, pp. 822-842
Open Access | Times Cited: 301
C9ORF72 poly(GA) aggregates sequester and impair HR23 and nucleocytoplasmic transport proteins
Yong‐Jie Zhang, Tania F. Gendron, Jonathan C. Grima, et al.
Nature Neuroscience (2016) Vol. 19, Iss. 5, pp. 668-677
Open Access | Times Cited: 300
Amyotrophic Lateral Sclerosis: An Update for 2018
Björn Oskarsson, Tania F. Gendron, Nathan P. Staff
Mayo Clinic Proceedings (2018) Vol. 93, Iss. 11, pp. 1617-1628
Open Access | Times Cited: 292
Poly(GR) impairs protein translation and stress granule dynamics in C9orf72-associated frontotemporal dementia and amyotrophic lateral sclerosis
Yong‐Jie Zhang, Tania F. Gendron, Mark Ebbert, et al.
Nature Medicine (2018) Vol. 24, Iss. 8, pp. 1136-1142
Open Access | Times Cited: 288
C9orf72 BAC Transgenic Mice Display Typical Pathologic Features of ALS/FTD
Jacqueline G. O’Rourke, Laurent Bogdanik, A.K.M. Ghulam Muhammad, et al.
Neuron (2015) Vol. 88, Iss. 5, pp. 892-901
Open Access | Times Cited: 284
Molecular mechanisms underlying nucleotide repeat expansion disorders
Indranil Malik, Chase P. Kelley, Eric T. Wang, et al.
Nature Reviews Molecular Cell Biology (2021) Vol. 22, Iss. 9, pp. 589-607
Open Access | Times Cited: 263
Human C9ORF72 Hexanucleotide Expansion Reproduces RNA Foci and Dipeptide Repeat Proteins but Not Neurodegeneration in BAC Transgenic Mice
Owen M. Peters, Gabriela Toro Cabrera, Hélène Tran, et al.
Neuron (2015) Vol. 88, Iss. 5, pp. 902-909
Open Access | Times Cited: 244
The Role of Dipeptide Repeats in C9ORF72-Related ALS-FTD
Brian D. Freibaum, J. Paul Taylor
Frontiers in Molecular Neuroscience (2017) Vol. 10
Open Access | Times Cited: 244
Disruption of RNA Metabolism in Neurological Diseases and Emerging Therapeutic Interventions
Julia K. Nussbacher, Ricardos Tabet, G Yeo, et al.
Neuron (2019) Vol. 102, Iss. 2, pp. 294-320
Open Access | Times Cited: 230
A C9ORF72/SMCR8-containing complex regulates ULK1 and plays a dual role in autophagy
Mei Yang, Liang Chen, Kunchithapadam Swaminathan, et al.
Science Advances (2016) Vol. 2, Iss. 9
Open Access | Times Cited: 225
C9orf72 Hexanucleotide Expansions Are Associated with Altered Endoplasmic Reticulum Calcium Homeostasis and Stress Granule Formation in Induced Pluripotent Stem Cell-Derived Neurons from Patients with Amyotrophic Lateral Sclerosis and Frontotemporal Dementia
Ruxandra Dafinca, Jakub Scaber, Nidaa A. Ababneh, et al.
Stem Cells (2016) Vol. 34, Iss. 8, pp. 2063-2078
Open Access | Times Cited: 223
Quantitative analysis and clinico-pathological correlations of different dipeptide repeat protein pathologies in C9ORF72 mutation carriers
Ian R. Mackenzie, Petra Frick, Friedrich A. Grässer, et al.
Acta Neuropathologica (2015) Vol. 130, Iss. 6, pp. 845-861
Closed Access | Times Cited: 219
Heterochromatin anomalies and double-stranded RNA accumulation underlie C9orf72 poly(PR) toxicity
Yong‐Jie Zhang, Lin Guo, Patrick Gonzales, et al.
Science (2019) Vol. 363, Iss. 6428
Open Access | Times Cited: 208
C9ORF72-ALS/FTD-associated poly(GR) binds Atp5a1 and compromises mitochondrial function in vivo
So Yoen Choi, Rodrigo López‐González, Gopinath Krishnan, et al.
Nature Neuroscience (2019) Vol. 22, Iss. 6, pp. 851-862
Open Access | Times Cited: 202
Regulation of mRNA Translation in Neurons—A Matter of Life and Death
Mridu Kapur, Caitlin E. Monaghan, Susan L. Ackerman
Neuron (2017) Vol. 96, Iss. 3, pp. 616-637
Open Access | Times Cited: 190
Differential Toxicity of Nuclear RNA Foci versus Dipeptide Repeat Proteins in a Drosophila Model of C9ORF72 FTD/ALS
Hélène Tran, Sandra Almeida, Jill E. Moore, et al.
Neuron (2015) Vol. 87, Iss. 6, pp. 1207-1214
Open Access | Times Cited: 187
The expanding biology of the C9orf72 nucleotide repeat expansion in neurodegenerative disease
Aaron R. Haeusler, Christopher J. Donnelly, Jeffrey D. Rothstein
Nature reviews. Neuroscience (2016) Vol. 17, Iss. 6, pp. 383-395
Open Access | Times Cited: 183
ALS: A bucket of genes, environment, metabolism and unknown ingredients
Mónica Zufiría, Francisco J. Gil‐Bea, Roberto Fernández‐Torrón, et al.
Progress in Neurobiology (2016) Vol. 142, pp. 104-129
Closed Access | Times Cited: 183
C9orf72 Poly(PR) Dipeptide Repeats Disturb Biomolecular Phase Separation and Disrupt Nucleolar Function
Michael R. White, Diana M. Mitrea, Peipei Zhang, et al.
Molecular Cell (2019) Vol. 74, Iss. 4, pp. 713-728.e6
Open Access | Times Cited: 174
p53 is a central regulator driving neurodegeneration caused by C9orf72 poly(PR)
Maya Maor-Nof, Zohar Shipony, Rodrigo López‐González, et al.
Cell (2021) Vol. 184, Iss. 3, pp. 689-708.e20
Open Access | Times Cited: 141
Suppression of mutant C9orf72 expression by a potent mixed backbone antisense oligonucleotide
Hélène Tran, Michael P. Moazami, Huiya Yang, et al.
Nature Medicine (2021) Vol. 28, Iss. 1, pp. 117-124
Open Access | Times Cited: 137
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