OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Mitochondrial fatty acid oxidation drives senescence
S. Yamauchi, Yuki Sugiura, Jon Yamaguchi, et al.
Science Advances (2024) Vol. 10, Iss. 43
Open Access | Times Cited: 6

Showing 6 citing articles:

Hypoxia-related immune subsets induced by Salmonella Typhi infection link early bacterial gut invasion to human infection outcomes
Noa Bossel Ben‐Moshe, Shelly Hen Avivi, Liron Levy Efrati, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2025)
Closed Access

Tissue-specific effects of dietary protein on cellular senescence are mediated by branched-chain amino acids
Mariah F. Calubag, Ismail Ademi, Isaac Grunow, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2025)
Open Access

Analysis and identification of mitochondria-related genes associated with age-related hearing loss
Tianyu Ma, Xiaoyun Zeng, Mengting Liu, et al.
BMC Genomics (2025) Vol. 26, Iss. 1
Open Access

Lactylation: A Novel Epigenetic Regulator of Cellular Senescence
Caiyu Sun, Jiaxuan Li, Lei Dong, et al.
Aging and Disease (2025)
Open Access

JNK Acts as A Conserved Regulator Prevents Healthy Aging by Attenuating Proteostasis Stress During Aging
Dan Xu, Wenxiang Yi, Haiying Fu, et al.
Research Square (Research Square) (2025)
Closed Access

Fatty acid oxidation contributed to NLRP3 inflammasome activation caused by N-nitrosamines co-exposure
Hu Zhang, Chao Zhao, Hong Guo, et al.
Food and Chemical Toxicology (2025), pp. 115549-115549
Closed Access

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