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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

A recessive form of hyper-IgE syndrome by disruption of ZNF341-dependent STAT3 transcription and activity
Vivien Béziat, Juan Li, Jian‐Xin Lin, et al.
Science Immunology (2018) Vol. 3, Iss. 24
Open Access | Times Cited: 175

Showing 1-25 of 175 citing articles:

Human Inborn Errors of Immunity: 2019 Update on the Classification from the International Union of Immunological Societies Expert Committee
Stuart G. Tangye, Waleed Al‐Herz, Aziz Bousfiha, et al.
Journal of Clinical Immunology (2020) Vol. 40, Iss. 1, pp. 24-64
Open Access | Times Cited: 1027
Human inborn errors of immunity: An expanding universe
Luigi D. Notarangelo, Rosa Bacchetta, Jean‐Laurent Casanova, et al.
Science Immunology (2020) Vol. 5, Iss. 49
Open Access | Times Cited: 206
Loss of the interleukin-6 receptor causes immunodeficiency, atopy, and abnormal inflammatory responses
Sarah Spencer, Sevgi Köstel Bal, William Egner, et al.
The Journal of Experimental Medicine (2019) Vol. 216, Iss. 9, pp. 1986-1998
Open Access | Times Cited: 181
Human IFN-γ immunity to mycobacteria is governed by both IL-12 and IL-23
Rubén Martínez‐Barricarte, Janet Markle, S. Cindy, et al.
Science Immunology (2018) Vol. 3, Iss. 30
Open Access | Times Cited: 165
Human autoantibodies underlying infectious diseases
Anne Puel, Paul Bastard, Jacinta Bustamante, et al.
The Journal of Experimental Medicine (2022) Vol. 219, Iss. 4
Open Access | Times Cited: 96
Human germline heterozygous gain-of-functionSTAT6variants cause severe allergic disease
Mehul Sharma, Daniel Leung, Mana Momenilandi, et al.
The Journal of Experimental Medicine (2023) Vol. 220, Iss. 5
Open Access | Times Cited: 63
JAKs and STATs from a Clinical Perspective: Loss-of-Function Mutations, Gain-of-Function Mutations, and Their Multidimensional Consequences
Nils Ott, Laura Faletti, Maximilian Heeg, et al.
Journal of Clinical Immunology (2023) Vol. 43, Iss. 6, pp. 1326-1359
Open Access | Times Cited: 43
Human STAT1 Gain-of-Function Heterozygous Mutations: Chronic Mucocutaneous Candidiasis and Type I Interferonopathy
Satoshi Okada, Takaki Asano, Kunihiko Moriya, et al.
Journal of Clinical Immunology (2020) Vol. 40, Iss. 8, pp. 1065-1081
Open Access | Times Cited: 127
Hyper IgE syndromes: clinical and molecular characteristics
Taha Al‐Shaikhly, Hans D. Ochs
Immunology and Cell Biology (2018) Vol. 97, Iss. 4, pp. 368-379
Open Access | Times Cited: 113
Lethal Infectious Diseases as Inborn Errors of Immunity: Toward a Synthesis of the Germ and Genetic Theories
Jean‐Laurent Casanova, Laurent Abel
Annual Review of Pathology Mechanisms of Disease (2020) Vol. 16, Iss. 1, pp. 23-50
Open Access | Times Cited: 106
STAT3 Hyper-IgE Syndrome—an Update and Unanswered Questions
Christo Tsilifis, Alexandra F. Freeman, Andrew R. Gennery
Journal of Clinical Immunology (2021) Vol. 41, Iss. 5, pp. 864-880
Open Access | Times Cited: 101
Regulation of the germinal center and humoral immunity by interleukin-21
Stuart G. Tangye, S. Cindy
The Journal of Experimental Medicine (2019) Vol. 217, Iss. 1
Open Access | Times Cited: 94
Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome
Vivien Béziat, Simon J. Tavernier, Yin‐Huai Chen, et al.
The Journal of Experimental Medicine (2020) Vol. 217, Iss. 6
Open Access | Times Cited: 92
Human inborn errors of immunity underlying superficial or invasive candidiasis
Anne Puel
Human Genetics (2020) Vol. 139, Iss. 6-7, pp. 1011-1022
Open Access | Times Cited: 82
Autoantibodies against cytokines: phenocopies of primary immunodeficiencies?
Cheng‐Lung Ku, Francesco Bianchi-Demicheli, Horst von Bernuth, et al.
Human Genetics (2020) Vol. 139, Iss. 6-7, pp. 783-794
Open Access | Times Cited: 81
STAT3 is critical for skeletal development and bone homeostasis by regulating osteogenesis
Siru Zhou, Qinggang Dai, Xiangru Huang, et al.
Nature Communications (2021) Vol. 12, Iss. 1
Open Access | Times Cited: 66
Human OTULIN haploinsufficiency impairs cell-intrinsic immunity to staphylococcal α-toxin
András N. Spaan, Anna‐Lena Neehus, Emmanuel Laplantine, et al.
Science (2022) Vol. 376, Iss. 6599
Open Access | Times Cited: 44
Impaired IL-23–dependent induction of IFN-γ underlies mycobacterial disease in patients with inherited TYK2 deficiency
Masato Ogishi, Andrés A. Arias, Rui Yang, et al.
The Journal of Experimental Medicine (2022) Vol. 219, Iss. 10
Open Access | Times Cited: 42
Interleukin‐21 in autoimmune and inflammatory skin diseases
Alberto Mesas‐Fernández, Euna Bodner, Franz J. Hilke, et al.
European Journal of Immunology (2023) Vol. 53, Iss. 4
Open Access | Times Cited: 30
Understanding the development of Th2 cell-driven allergic airway disease in early life
Beatriz León
Frontiers in Allergy (2023) Vol. 3
Open Access | Times Cited: 29
Inborn errors of human B cell development, differentiation, and function
Stuart G. Tangye, Tina Nguyen, Elissa K. Deenick, et al.
The Journal of Experimental Medicine (2023) Vol. 220, Iss. 7
Open Access | Times Cited: 26
IL-6 prevents Th2 cell polarization by promoting SOCS3-dependent suppression of IL-2 signaling
Holly Bachus, Erin McLaughlin, Crystal Lewis, et al.
Cellular and Molecular Immunology (2023) Vol. 20, Iss. 6, pp. 651-665
Open Access | Times Cited: 23
FLT3L governs the development of partially overlapping hematopoietic lineages in humans and mice
Mana Momenilandi, Romain Lévy, Steicy Sobrino, et al.
Cell (2024) Vol. 187, Iss. 11, pp. 2817-2837.e31
Open Access | Times Cited: 13
The genetics of hyper IgE syndromes
Randa S AlYafie, Dinesh Velayutham, Nicholas van Panhuys, et al.
Frontiers in Immunology (2025) Vol. 16
Open Access | Times Cited: 1
Selective loss of function variants in IL6ST cause Hyper-IgE syndrome with distinct impairments of T-cell phenotype and function
Tala Shahin, Dominik Aschenbrenner, Deniz Çağdaş, et al.
Haematologica (2018) Vol. 104, Iss. 3, pp. 609-621
Open Access | Times Cited: 83
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