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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Huntingtin promotes mTORC1 signaling in the pathogenesis of Huntington’s disease
William M. Pryor, Marta Biagioli, Neelam Shahani, et al.
Science Signaling (2014) Vol. 7, Iss. 349
Closed Access | Times Cited: 115

Showing 1-25 of 115 citing articles:

Regulation and metabolic functions of mTORC1 and mTORC2
Angelia Szwed, Eugene Kim, Estela Jacinto
Physiological Reviews (2021) Vol. 101, Iss. 3, pp. 1371-1426
Open Access | Times Cited: 484
mTOR in Brain Physiology and Pathologies
Joël Bockaert, Philippe Marin
Physiological Reviews (2015) Vol. 95, Iss. 4, pp. 1157-1187
Closed Access | Times Cited: 320
The mTOR signalling cascade: paving new roads to cure neurological disease
Peter B. Crino
Nature Reviews Neurology (2016) Vol. 12, Iss. 7, pp. 379-392
Closed Access | Times Cited: 312
Role of metabolism in neurodegenerative disorders
Claudio Procaccini, Marianna Santopaolo, Deriggio Faicchia, et al.
Metabolism (2016) Vol. 65, Iss. 9, pp. 1376-1390
Closed Access | Times Cited: 202
Neurodegenerative Diseases: Regenerative Mechanisms and Novel Therapeutic Approaches
Rashad Hussain, Hira Zubair, Sarah Pursell, et al.
Brain Sciences (2018) Vol. 8, Iss. 9, pp. 177-177
Open Access | Times Cited: 194
Mammalian/mechanistic target of rapamycin (mTOR) complexes in neurodegeneration
Henry Querfurth, Hankyu Lee
Molecular Neurodegeneration (2021) Vol. 16, Iss. 1
Open Access | Times Cited: 186
Autophagic and endo-lysosomal dysfunction in neurodegenerative disease
Bilal R. Malik, Daniel C. Maddison, Gaynor A. Smith, et al.
Molecular Brain (2019) Vol. 12, Iss. 1
Open Access | Times Cited: 158
Gene therapy for neurodegenerative disorders: advances, insights and prospects
Wei Chen, Yang Hu, Dianwen Ju
Acta Pharmaceutica Sinica B (2020) Vol. 10, Iss. 8, pp. 1347-1359
Open Access | Times Cited: 158
Therapeutic Advances for Huntington’s Disease
Ashok Kumar, Vijay Kumar, Kritanjali Singh, et al.
Brain Sciences (2020) Vol. 10, Iss. 1, pp. 43-43
Open Access | Times Cited: 142
Targeting molecules to medicine with mTOR, autophagy and neurodegenerative disorders
Kenneth Maiese
British Journal of Clinical Pharmacology (2015) Vol. 82, Iss. 5, pp. 1245-1266
Open Access | Times Cited: 171
Fatty acid metabolism in the progression and resolution of CNS disorders
Jeroen F. J. Bogie, Mansour Haidar, Gijs Kooij, et al.
Advanced Drug Delivery Reviews (2020) Vol. 159, pp. 198-213
Open Access | Times Cited: 126
Cyclic GMP-AMP synthase promotes the inflammatory and autophagy responses in Huntington disease
Manish Sharma, Sumitha Rajendrarao, Neelam Shahani, et al.
Proceedings of the National Academy of Sciences (2020) Vol. 117, Iss. 27, pp. 15989-15999
Open Access | Times Cited: 113
How Lysosomes Sense, Integrate, and Cope with Stress
Paul Säftig, Rosa Puertollano
Trends in Biochemical Sciences (2020) Vol. 46, Iss. 2, pp. 97-112
Open Access | Times Cited: 112
mTOR/AMPK signaling in the brain: Cell metabolism, proteostasis and survival
Carla Garza-Lombó, Annika Schroder, Elsa M. Reyes‐Reyes, et al.
Current Opinion in Toxicology (2018) Vol. 8, pp. 102-110
Open Access | Times Cited: 100
mTOR-Related Brain Dysfunctions in Neuropsychiatric Disorders
Larisa Ryskalin, Fiona Limanaqi, Alessandro Frati, et al.
International Journal of Molecular Sciences (2018) Vol. 19, Iss. 8, pp. 2226-2226
Open Access | Times Cited: 96
Mutant Huntingtin stalls ribosomes and represses protein synthesis in a cellular model of Huntington disease
Mehdi Eshraghi, Pabalu P. Karunadharma, Juliana Blin, et al.
Nature Communications (2021) Vol. 12, Iss. 1
Open Access | Times Cited: 90
CRISPR/Cas9 Mediated Therapeutic Approach in Huntington’s Disease
Süleyman Serdar Alkanlı, Nevra Alkanlı, Arzu Ay, et al.
Molecular Neurobiology (2022) Vol. 60, Iss. 3, pp. 1486-1498
Open Access | Times Cited: 45
Hydrogen sulfide signalling in neurodegenerative diseases
Sunil Jamuna Tripathi, Suwarna Chakraborty, Emiko Miller, et al.
British Journal of Pharmacology (2023)
Closed Access | Times Cited: 25
mTOR inhibition in Q175 Huntington’s disease model mice facilitates neuronal autophagy and mutant huntingtin clearance
Philip Stavrides, Chris N. Goulbourne, James Peddy, et al.
(2025)
Open Access | Times Cited: 1
The Ras Superfamily of Small GTPases in Non-neoplastic Cerebral Diseases
Liang Qu, Chao Pan, Shiming He, et al.
Frontiers in Molecular Neuroscience (2019) Vol. 12
Open Access | Times Cited: 76
Activation of AMPK-induced autophagy ameliorates Huntington disease pathology in vitro
Carolin Walter, Laura E. Clemens, Amelie J. Müller, et al.
Neuropharmacology (2016) Vol. 108, pp. 24-38
Closed Access | Times Cited: 71
Autophagy—from molecular mechanisms to clinical relevance
Mónika Lippai, Zsuzsanna Szatmári
Cell Biology and Toxicology (2016) Vol. 33, Iss. 2, pp. 145-168
Closed Access | Times Cited: 69
Rhes travels from cell to cell and transports Huntington disease protein via TNT-like protrusion
Manish Sharma, Srinivasa Subramaniam
The Journal of Cell Biology (2019) Vol. 218, Iss. 6, pp. 1972-1993
Open Access | Times Cited: 64
Increased translation as a novel pathogenic mechanism in Huntington’s disease
Jordi Creus‐Muncunill, Raquel Badillos-Rodríguez, Marta Garcia‐Forn, et al.
Brain (2019) Vol. 142, Iss. 10, pp. 3158-3175
Open Access | Times Cited: 55
Deletion of SUMO1 attenuates behavioral and anatomical deficits by regulating autophagic activities in Huntington disease
Uri Nimrod Ramírez-Jarquín, Manish Sharma, Wuyue Zhou, et al.
Proceedings of the National Academy of Sciences (2022) Vol. 119, Iss. 5
Open Access | Times Cited: 29
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