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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Targeting RNA Foci in iPSC-Derived Motor Neurons from ALS Patients with a C9ORF72 Repeat Expansion
Dhruv Sareen, Jacqueline G. O’Rourke, Pratap Meera, et al.
Science Translational Medicine (2013) Vol. 5, Iss. 208
Open Access | Times Cited: 634

Showing 1-25 of 634 citing articles:

Decoding ALS: from genes to mechanism
J. Paul Taylor, Robert H. Brown, Don W. Cleveland
Nature (2016) Vol. 539, Iss. 7628, pp. 197-206
Open Access | Times Cited: 1821
Amyotrophic Lateral Sclerosis
Robert H. Brown, Ammar Al‐Chalabi
New England Journal of Medicine (2017) Vol. 377, Iss. 2, pp. 162-172
Open Access | Times Cited: 1704
Blood-Brain Barrier: From Physiology to Disease and Back
Melanie D. Sweeney, Zhen Zhao, Axel Montagne, et al.
Physiological Reviews (2018) Vol. 99, Iss. 1, pp. 21-78
Open Access | Times Cited: 1674
C9orf72 nucleotide repeat structures initiate molecular cascades of disease
Aaron R. Haeusler, Christopher J. Donnelly, Goran Periz, et al.
Nature (2014) Vol. 507, Iss. 7491, pp. 195-200
Open Access | Times Cited: 921
The C9orf72 repeat expansion disrupts nucleocytoplasmic transport
Ke Zhang, Christopher J. Donnelly, Aaron R. Haeusler, et al.
Nature (2015) Vol. 525, Iss. 7567, pp. 56-61
Open Access | Times Cited: 920
GGGGCC repeat expansion in C9orf72 compromises nucleocytoplasmic transport
Brian D. Freibaum, Yubing Lu, Rodrigo López‐González, et al.
Nature (2015) Vol. 525, Iss. 7567, pp. 129-133
Open Access | Times Cited: 791
C9orf72 repeat expansions cause neurodegeneration in Drosophila through arginine-rich proteins
Sarah Mizielinska, Sebastian Grönke, Teresa Niccoli, et al.
Science (2014) Vol. 345, Iss. 6201, pp. 1192-1194
Open Access | Times Cited: 695
ALS Genetics, Mechanisms, and Therapeutics: Where Are We Now?
Rita Mejzini, Loren L. Flynn, Ianthe Pitout, et al.
Frontiers in Neuroscience (2019) Vol. 13
Open Access | Times Cited: 655
Intrinsic Membrane Hyperexcitability of Amyotrophic Lateral Sclerosis Patient-Derived Motor Neurons
Brian J. Wainger, Evangelos Kiskinis, Cassidy Mellin, et al.
Cell Reports (2014) Vol. 7, Iss. 1, pp. 1-11
Open Access | Times Cited: 619
C9orf72-mediated ALS and FTD: multiple pathways to disease
Rubika Balendra, Adrian M. Isaacs
Nature Reviews Neurology (2018) Vol. 14, Iss. 9, pp. 544-558
Open Access | Times Cited: 615
Non-coding genetic variants in human disease: Figure 1.
Feng Zhang, James R. Lupski
Human Molecular Genetics (2015) Vol. 24, Iss. R1, pp. R102-R110
Open Access | Times Cited: 568
Pluripotent stem cells in disease modelling and drug discovery
Yishai Avior, Ido Sagi, Nissim Benvenisty
Nature Reviews Molecular Cell Biology (2016) Vol. 17, Iss. 3, pp. 170-182
Closed Access | Times Cited: 551
Haploinsufficiency leads to neurodegeneration in C9ORF72 ALS/FTD human induced motor neurons
Yingxiao Shi, Shao‐Yu Lin, Kim A. Staats, et al.
Nature Medicine (2018) Vol. 24, Iss. 3, pp. 313-325
Open Access | Times Cited: 519
C9orf72 is required for proper macrophage and microglial function in mice
Jacqueline G. O’Rourke, Laurent Bogdanik, Alberto Yáñez, et al.
Science (2016) Vol. 351, Iss. 6279, pp. 1324-1329
Open Access | Times Cited: 505
Gain of Toxicity from ALS/FTD-Linked Repeat Expansions in C9ORF72 Is Alleviated by Antisense Oligonucleotides Targeting GGGGCC-Containing RNAs
Jie Jiang, Qiang Zhu, Tania F. Gendron, et al.
Neuron (2016) Vol. 90, Iss. 3, pp. 535-550
Open Access | Times Cited: 502
C9ORF72 GGGGCC Expanded Repeats Produce Splicing Dysregulation which Correlates with Disease Severity in Amyotrophic Lateral Sclerosis
Johnathan Cooper‐Knock, Joanna J. Bury, Paul R. Heath, et al.
PLoS ONE (2015) Vol. 10, Iss. 5, pp. e0127376-e0127376
Open Access | Times Cited: 447
Pathways Disrupted in Human ALS Motor Neurons Identified through Genetic Correction of Mutant SOD1
Evangelos Kiskinis, Jackson Sandoe, Luis A. Williams, et al.
Cell stem cell (2014) Vol. 14, Iss. 6, pp. 781-795
Open Access | Times Cited: 427
Poly(GR) in C9ORF72 -Related ALS/FTD Compromises Mitochondrial Function and Increases Oxidative Stress and DNA Damage in iPSC-Derived Motor Neurons
Rodrigo López‐González, Yubing Lu, Tania F. Gendron, et al.
Neuron (2016) Vol. 92, Iss. 2, pp. 383-391
Open Access | Times Cited: 384
C9ORF72 repeat expansions in mice cause TDP-43 pathology, neuronal loss, and behavioral deficits
Jeannie Chew, Tania F. Gendron, Mercedes Prudencio, et al.
Science (2015) Vol. 348, Iss. 6239, pp. 1151-1154
Open Access | Times Cited: 371
Distinct brain transcriptome profiles in C9orf72-associated and sporadic ALS
Mercedes Prudencio, Véronique Belzil, Ranjan Batra, et al.
Nature Neuroscience (2015) Vol. 18, Iss. 8, pp. 1175-1182
Open Access | Times Cited: 363
The C9orf72 protein interacts with Rab1a and the ULK1 complex to regulate initiation of autophagy
Christopher P Webster, Emma F. Smith, Claudia S. Bauer, et al.
The EMBO Journal (2016) Vol. 35, Iss. 15, pp. 1656-1676
Open Access | Times Cited: 362
Microphysiological 3D model of amyotrophic lateral sclerosis (ALS) from human iPS-derived muscle cells and optogenetic motor neurons
Tatsuya Osaki, Sebastien G. M. Uzel, Roger D. Kamm
Science Advances (2018) Vol. 4, Iss. 10
Open Access | Times Cited: 351
The frontotemporal dementia-motor neuron disease continuum
James R. Burrell, Glenda M. Halliday, Jillian J. Kril, et al.
The Lancet (2016) Vol. 388, Iss. 10047, pp. 919-931
Closed Access | Times Cited: 344
Discovery of a Biomarker and Lead Small Molecules to Target r(GGGGCC)-Associated Defects in c9FTD/ALS
Zhaoming Su, Yong‐Jie Zhang, Tania F. Gendron, et al.
Neuron (2014) Vol. 83, Iss. 5, pp. 1043-1050
Open Access | Times Cited: 341
C9orf72 FTLD/ALS-associated Gly-Ala dipeptide repeat proteins cause neuronal toxicity and Unc119 sequestration
Stephanie May, Daniel Hornburg, Martin H. Schludi, et al.
Acta Neuropathologica (2014) Vol. 128, Iss. 4, pp. 485-503
Open Access | Times Cited: 310
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