OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Mechanisms Underlying Acute Protection From Cardiac Ischemia-Reperfusion Injury
Elizabeth Murphy, Charles Steenbergen
Physiological Reviews (2008) Vol. 88, Iss. 2, pp. 581-609
Open Access | Times Cited: 1359

Showing 1-25 of 1359 citing articles:

Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS
Edward T. Chouchani, Victoria R. Pell, Edoardo Gaude, et al.
Nature (2014) Vol. 515, Iss. 7527, pp. 431-435
Open Access | Times Cited: 2379

Lanthanide Nanoparticles: From Design toward Bioimaging and Therapy
Hao Dong, Shuoren Du, Xiaoyu Zheng, et al.
Chemical Reviews (2015) Vol. 115, Iss. 19, pp. 10725-10815
Closed Access | Times Cited: 1018

Fundamental Mechanisms of Regulated Cell Death and Implications for Heart Disease
Dominic P. Del Re, Dulguun Amgalan, Andreas Linkermann, et al.
Physiological Reviews (2019) Vol. 99, Iss. 4, pp. 1765-1817
Open Access | Times Cited: 746

Cell Death in the Pathogenesis of Heart Disease: Mechanisms and Significance
Russell S. Whelan, Vladimir Kaplinskiy, Richard N. Kitsis
Annual Review of Physiology (2010) Vol. 72, Iss. 1, pp. 19-44
Closed Access | Times Cited: 722

ROS and redox signaling in myocardial ischemia-reperfusion injury and cardioprotection
Susana Cadenas
Free Radical Biology and Medicine (2018) Vol. 117, pp. 76-89
Closed Access | Times Cited: 673

Mitochondrial dysfunction and oxidative stress in heart disease
Jessica N. Peoples, Anita Saraf, Nasab Ghazal, et al.
Experimental & Molecular Medicine (2019) Vol. 51, Iss. 12, pp. 1-13
Open Access | Times Cited: 661

Mitochondrial function as a therapeutic target in heart failure
David A. Brown, Justin B. Perry, Mitchell E. Allen, et al.
Nature Reviews Cardiology (2016) Vol. 14, Iss. 4, pp. 238-250
Open Access | Times Cited: 643

A Unifying Mechanism for Mitochondrial Superoxide Production during Ischemia-Reperfusion Injury
Edward T. Chouchani, Victoria R. Pell, Andrew M. James, et al.
Cell Metabolism (2016) Vol. 23, Iss. 2, pp. 254-263
Open Access | Times Cited: 623

Cardioprotection by S-nitrosation of a cysteine switch on mitochondrial complex I
Edward T. Chouchani, Carmen Methner, Sergiy M. Nadtochiy, et al.
Nature Medicine (2013) Vol. 19, Iss. 6, pp. 753-759
Open Access | Times Cited: 576

ROS and diseases: role in metabolism and energy supply
Shenshu Yang, Gaojian Lian
Molecular and Cellular Biochemistry (2019) Vol. 467, Iss. 1-2, pp. 1-12
Open Access | Times Cited: 503

S -Nitrosylation in Cardiovascular Signaling
Brian Lima, Michael T. Forrester, Douglas T. Hess, et al.
Circulation Research (2010) Vol. 106, Iss. 4, pp. 633-646
Open Access | Times Cited: 468

The mitochondrial permeability transition pore: Molecular nature and role as a target in cardioprotection
Paolo Bernardi, Fabio Di Lisa
Journal of Molecular and Cellular Cardiology (2014) Vol. 78, pp. 100-106
Open Access | Times Cited: 449

Cardioprotection
Gerd Heusch, Kerstin Boengler, Rainer Schulz
Circulation (2008) Vol. 118, Iss. 19, pp. 1915-1919
Closed Access | Times Cited: 434

Hexokinase II integrates energy metabolism and cellular protection: Akting on mitochondria and TORCing to autophagy
David J. Roberts, Shigeki Miyamoto
Cell Death and Differentiation (2014) Vol. 22, Iss. 2, pp. 248-257
Open Access | Times Cited: 423

The mitochondrial permeability transition: A current perspective on its identity and role in ischaemia/reperfusion injury
Andrew P. Halestrap, A. Richardson
Journal of Molecular and Cellular Cardiology (2014) Vol. 78, pp. 129-141
Closed Access | Times Cited: 410

The Opa1-Dependent Mitochondrial Cristae Remodeling Pathway Controls Atrophic, Apoptotic, and Ischemic Tissue Damage
Tatiana Varanita, María Eugenia Soriano, Vanina Romanello, et al.
Cell Metabolism (2015) Vol. 21, Iss. 6, pp. 834-844
Open Access | Times Cited: 401

Exposure to Hydrogen Peroxide Induces Oxidation and Activation of AMP-activated Protein Kinase*
Jaroslaw W. Zmijewski, Sami Banerjee, Hong‐Beom Bae, et al.
Journal of Biological Chemistry (2010) Vol. 285, Iss. 43, pp. 33154-33164
Open Access | Times Cited: 394

Physiological and Pathological Roles of the Mitochondrial Permeability Transition Pore in the Heart
Jennifer Q. Kwong, Jeffery D. Molkentin
Cell Metabolism (2015) Vol. 21, Iss. 2, pp. 206-214
Open Access | Times Cited: 391

MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca2+ overload and cell death
Arin B. Aurora, Ahmed I. Mahmoud, Xiang Luo, et al.
Journal of Clinical Investigation (2012) Vol. 122, Iss. 4, pp. 1222-1232
Open Access | Times Cited: 369

Role of Cardiolipin in Mitochondrial Function and Dynamics in Health and Disease: Molecular and Pharmacological Aspects
Giuseppe Paradies, Valeria Paradies, Francesca Ruggiero, et al.
Cells (2019) Vol. 8, Iss. 7, pp. 728-728
Open Access | Times Cited: 363

Spliced X-Box Binding Protein 1 Couples the Unfolded Protein Response to Hexosamine Biosynthetic Pathway
Zhao V. Wang, Yingfeng Deng, Ningguo Gao, et al.
Cell (2014) Vol. 156, Iss. 6, pp. 1179-1192
Open Access | Times Cited: 361

Mitochondrial Dysfunction and Myocardial Ischemia-Reperfusion: Implications for Novel Therapies
Edward J. Lesnefsky, Qun Chen, Bernard Tandler, et al.
The Annual Review of Pharmacology and Toxicology (2016) Vol. 57, Iss. 1, pp. 535-565
Closed Access | Times Cited: 359

The role of the mitochondrial permeability transition pore in heart disease
Andrew P. Halestrap, Philippe Pasdois
Biochimica et Biophysica Acta (BBA) - Bioenergetics (2009) Vol. 1787, Iss. 11, pp. 1402-1415
Closed Access | Times Cited: 354

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