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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Mitochondrial Determinants of Doxorubicin-Induced Cardiomyopathy
Kendall B. Wallace, Vilma A. Sardão, Paulo J. Oliveira
Circulation Research (2020) Vol. 126, Iss. 7, pp. 926-941
Open Access | Times Cited: 424

Showing 1-25 of 424 citing articles:

PRMT4 promotes ferroptosis to aggravate doxorubicin-induced cardiomyopathy via inhibition of the Nrf2/GPX4 pathway
Yilong Wang, Shu Yan, Xuemei Liu, et al.
Cell Death and Differentiation (2022) Vol. 29, Iss. 10, pp. 1982-1995
Open Access | Times Cited: 212
Meteorin-like protein attenuates doxorubicin-induced cardiotoxicity via activating cAMP/PKA/SIRT1 pathway
Can Hu, Xin Zhang, Peng Song, et al.
Redox Biology (2020) Vol. 37, pp. 101747-101747
Open Access | Times Cited: 183
Mitochondrial quality control mechanisms as therapeutic targets in doxorubicin-induced cardiotoxicity
Lin Wu, Litao Wang, Yuxin Du, et al.
Trends in Pharmacological Sciences (2022) Vol. 44, Iss. 1, pp. 34-49
Closed Access | Times Cited: 137
Cooperative sensing of mitochondrial DNA by ZBP1 and cGAS promotes cardiotoxicity
Yuanjiu Lei, Jordyn J. VanPortfliet, Yifan Chen, et al.
Cell (2023) Vol. 186, Iss. 14, pp. 3013-3032.e22
Open Access | Times Cited: 123
Mitochondrial-Targeted Therapy for Doxorubicin-Induced Cardiotoxicity
Bin Bin Wu, Kam Tong Leung, Ellen Poon
International Journal of Molecular Sciences (2022) Vol. 23, Iss. 3, pp. 1912-1912
Open Access | Times Cited: 122
Analysis of Models of Doxorubicin-Induced Cardiomyopathy in Rats and Mice. A Modern View From the Perspective of the Pathophysiologist and the Clinician
Ekaterina Podyacheva, Е. А. Кушнарева, А. А. Карпов, et al.
Frontiers in Pharmacology (2021) Vol. 12
Open Access | Times Cited: 107
Therapeutic Targets for DOX-Induced Cardiomyopathy: Role of Apoptosis vs. Ferroptosis
Hiroki Kitakata, Jin Endo, Hidehiko Ikura, et al.
International Journal of Molecular Sciences (2022) Vol. 23, Iss. 3, pp. 1414-1414
Open Access | Times Cited: 101
Drug-induced oxidative stress in cancer treatments: Angel or devil?
Hao Jiang, Jing Zuo, Bowen Li, et al.
Redox Biology (2023) Vol. 63, pp. 102754-102754
Open Access | Times Cited: 100
Mitochondrial outer membrane protein FUNDC2 promotes ferroptosis and contributes to doxorubicin-induced cardiomyopathy
Na Ta, Chuanren Qu, Hao Wu, et al.
Proceedings of the National Academy of Sciences (2022) Vol. 119, Iss. 36
Open Access | Times Cited: 99
Cardiotoxicity of Anticancer Drugs: Molecular Mechanisms and Strategies for Cardioprotection
Marco Bruno Morelli, Chiara Bongiovanni, Silvia Da Pra, et al.
Frontiers in Cardiovascular Medicine (2022) Vol. 9
Open Access | Times Cited: 95
Dilated cardiomyopathy: causes, mechanisms, and current and future treatment approaches
Stéphane Heymans, Neal K. Lakdawala, Carsten Tschöpe, et al.
The Lancet (2023) Vol. 402, Iss. 10406, pp. 998-1011
Closed Access | Times Cited: 81
Environmental Chemical Exposures and Mitochondrial Dysfunction: a Review of Recent Literature
Aalekhya Reddam, Sarah M. McLarnan, Allison Kupsco
Current Environmental Health Reports (2022) Vol. 9, Iss. 4, pp. 631-649
Open Access | Times Cited: 74
Critical Role of the cGAS-STING Pathway in Doxorubicin-Induced Cardiotoxicity
Wei Luo, Xiaoyi Zou, Yidan Wang, et al.
Circulation Research (2023) Vol. 132, Iss. 11
Open Access | Times Cited: 73
Molecular insights into the pathophysiology of doxorubicin-induced cardiotoxicity: a graphical representation
Nonhlakanipho F. Sangweni, Kwazi Gabuza, Barbara Huisamen, et al.
Archives of Toxicology (2022) Vol. 96, Iss. 6, pp. 1541-1550
Open Access | Times Cited: 70
Inhibition of METTL3 ameliorates doxorubicin-induced cardiotoxicity through suppression of TFRC-mediated ferroptosis
Lin Wu, Yuxin Du, Litao Wang, et al.
Redox Biology (2024) Vol. 72, pp. 103157-103157
Open Access | Times Cited: 21
Cellular Senescence, Mitochondrial Dysfunction, and Their Link to Cardiovascular Disease
María Camacho Encina, Laura Booth, Rachael Redgrave, et al.
Cells (2024) Vol. 13, Iss. 4, pp. 353-353
Open Access | Times Cited: 18
Targeted pH- and redox-responsive AuS/micelles with low CMC for highly efficient sonodynamic therapy of metastatic breast cancer
Samane Maghsoudian, Hamidreza Motasadizadeh, Hassan Farhadnejad, et al.
Biomaterials Advances (2024) Vol. 158, pp. 213771-213771
Closed Access | Times Cited: 16
6PPD induced cardiac dysfunction in zebrafish associated with mitochondrial damage and inhibition of autophagy processes
Chanlin Fang, Shanshan Di, Yundong Yu, et al.
Journal of Hazardous Materials (2024) Vol. 471, pp. 134357-134357
Closed Access | Times Cited: 16
Rnd3 protects against doxorubicin-induced cardiotoxicity through inhibition of PANoptosis in a Rock1/Drp1/mitochondrial fission-dependent manner
Ge Wen, Xiaohua Zhang, Jie Lin, et al.
Cell Death and Disease (2025) Vol. 16, Iss. 1
Open Access | Times Cited: 2
DNA Repair Pathways in the Mitochondria
Dillon E. King, William C. Copeland
DNA repair (2025) Vol. 146, pp. 103814-103814
Closed Access | Times Cited: 2
The mitochondrial permeability transition pore: an evolving concept critical for cell life and death
Giampaolo Morciano, Natalia Naumova, Piotr Koprowski, et al.
Biological reviews/Biological reviews of the Cambridge Philosophical Society (2021) Vol. 96, Iss. 6, pp. 2489-2521
Closed Access | Times Cited: 89
Research progress of therapeutic drugs for doxorubicin-induced cardiomyopathy
Ye Chen‐Izu, Saixian Shi, Yan Dai
Biomedicine & Pharmacotherapy (2022) Vol. 156, pp. 113903-113903
Open Access | Times Cited: 69
Mitochondrial Dysfunction Is a Common Denominator Linking Skeletal Muscle Wasting Due to Disease, Aging, and Prolonged Inactivity
Hayden W. Hyatt, Scott K. Powers
Antioxidants (2021) Vol. 10, Iss. 4, pp. 588-588
Open Access | Times Cited: 68
Mitochondria-Rich Extracellular Vesicles Rescue Patient-Specific Cardiomyocytes From Doxorubicin Injury
Connor O’Brien, Mehmet Ozgün Ozen, Gentaro Ikeda, et al.
JACC CardioOncology (2021) Vol. 3, Iss. 3, pp. 428-440
Open Access | Times Cited: 68
Tanshinone I inhibits doxorubicin-induced cardiotoxicity by regulating Nrf2 signaling pathway
Qianqian Jiang, Xu Chen, Xue Tian, et al.
Phytomedicine (2022) Vol. 106, pp. 154439-154439
Closed Access | Times Cited: 67
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