OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

KrasP34R and KrasT58I mutations induce distinct RASopathy phenotypes in mice
Jasmine C. Wong, Pedro A. Pérez–Mancera, Tannie Q. Huang, et al.
JCI Insight (2020) Vol. 5, Iss. 21
Open Access | Times Cited: 15

Showing 15 citing articles:

The RASopathies: from pathogenetics to therapeutics
Katie E. Hebron, Edjay R. Hernandez, Marielle E. Yohe
Disease Models & Mechanisms (2022) Vol. 15, Iss. 2
Open Access | Times Cited: 117

What Causes Hypertrophic Cardiomyopathy?
Bradley A. Maron, Rui‐Sheng Wang, Mercedes R. Carnethon, et al.
The American Journal of Cardiology (2022) Vol. 179, pp. 74-82
Open Access | Times Cited: 31

New prospectives on treatment opportunities in RASopathies
Bruce D. Gelb, Marielle E. Yohe, Cordula M. Wolf, et al.
American Journal of Medical Genetics Part C Seminars in Medical Genetics (2022) Vol. 190, Iss. 4, pp. 541-560
Open Access | Times Cited: 24

Impaired Proteolysis of Noncanonical RAS Proteins Drives Clonal Hematopoietic Transformation
Sisi Chen, Rahul S. Vedula, Antonio Cuevas-Navarro, et al.
Cancer Discovery (2022) Vol. 12, Iss. 10, pp. 2434-2453
Open Access | Times Cited: 23

MEK Inhibition for RASopathy-Associated Hypertrophic Cardiomyopathy: Clinical Application of a Basic Concept
Dominic Chaput, Grégor Andelfinger
Canadian Journal of Cardiology (2024) Vol. 40, Iss. 5, pp. 789-799
Closed Access | Times Cited: 5

Missense and truncated variants in ERF in individuals with a Noonan-like phenotype without craniosynostosis
Yusuke Goto, Tetsuya Niihori, Seiji Mizuno, et al.
Scientific Reports (2025) Vol. 15, Iss. 1
Open Access

RASopathies: The musculoskeletal consequences and their etiology and pathogenesis
John L. Fowlkes, Kathryn M. Thrailkill, R. Clay Bunn
Bone (2021) Vol. 152, pp. 116060-116060
Open Access | Times Cited: 18

Defining the spatial landscape of KRAS mutated congenital pulmonary airway malformations: a distinct entity with a spectrum of histopathologic features
Nya D. Nelson, Feng Xu, Prashant Chandrasekaran, et al.
Modern Pathology (2022) Vol. 35, Iss. 12, pp. 1870-1881
Open Access | Times Cited: 13

Combined HRAS and NRAS ablation induces a RASopathy phenotype in mice
Rocío Fuentes‐Mateos, Rósula García‐Navas, Cristina Fernández‐Infante, et al.
Cell Communication and Signaling (2024) Vol. 22, Iss. 1
Open Access

Craniofacial and Dental Development in the RASopathies
Kaitlin Katsura, Andrew H. Jheon, Alice F. Goodwin
(2024), pp. 757-782
Closed Access

Modeling the Non-NF1 RASopathies
Christine Insinna, Marielle E. Yohe
(2024), pp. 869-893
Closed Access

Single cell proteomics characterization of bone marrow hematopoiesis with distinct Ras pathway lesions
Laila Karra, Anna‐Marie Finger, Lauren A. Shechtman, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2023)
Open Access | Times Cited: 1

Emerging Hypotheses on the Pathogenesis of Rasopathy-Associated Cardiomyopathies
Shun Nakagama, Masataka Yamasaki, Masamichi Ito, et al.
Pediatric Cardiology and Cardiac Surgery (2023) Vol. 39, Iss. 4, pp. 192-199
Open Access

RAS mutation patterns arise from tissue-specific responses to distinct oncogenic signaling
Özgün Erdoğan, Nicole L Pershing, Erin Kaltenbrun, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2021)
Open Access

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