OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Cellular and molecular mechanisms in kidney fibrosis
Jeremy S. Duffield
Journal of Clinical Investigation (2014) Vol. 124, Iss. 6, pp. 2299-2306
Open Access | Times Cited: 555

Showing 1-25 of 555 citing articles:

Cardiac Fibrosis
Joshua G. Travers, Fadia Kamal, Jeffrey Robbins, et al.
Circulation Research (2016) Vol. 118, Iss. 6, pp. 1021-1040
Open Access | Times Cited: 1338

Perivascular Gli1+ Progenitors Are Key Contributors to Injury-Induced Organ Fibrosis
Rafael Kramann, Rebekka K. Schneider, Derek P. DiRocco, et al.
Cell stem cell (2014) Vol. 16, Iss. 1, pp. 51-66
Open Access | Times Cited: 821

Failed Tubule Recovery, AKI-CKD Transition, and Kidney Disease Progression
Manjeri A. Venkatachalam, Joel M. Weinberg, Wilhelm Kriz, et al.
Journal of the American Society of Nephrology (2015) Vol. 26, Iss. 8, pp. 1765-1776
Open Access | Times Cited: 632

FGF/FGFR signaling in health and disease
Yangli Xie, Nan Su, Jing Yang, et al.
Signal Transduction and Targeted Therapy (2020) Vol. 5, Iss. 1
Open Access | Times Cited: 600

Decoding myofibroblast origins in human kidney fibrosis
Christoph Kuppe, Mahmoud M. Ibrahim, Jennifer Kranz, et al.
Nature (2020) Vol. 589, Iss. 7841, pp. 281-286
Open Access | Times Cited: 539

The sirtuin family in health and disease
Qi‐Jun Wu, Tie‐Ning Zhang, Huanhuan Chen, et al.
Signal Transduction and Targeted Therapy (2022) Vol. 7, Iss. 1
Open Access | Times Cited: 415

Cellular and molecular mechanisms of kidney fibrosis
Sonja Djudjaj, Peter Boor
Molecular Aspects of Medicine (2018) Vol. 65, pp. 16-36
Closed Access | Times Cited: 382

Anti–microRNA-21 oligonucleotides prevent Alport nephropathy progression by stimulating metabolic pathways
Ivan G. Gomez, Deidre A. MacKenna, Bryce G. Johnson, et al.
Journal of Clinical Investigation (2014) Vol. 125, Iss. 1, pp. 141-156
Open Access | Times Cited: 378

Mesenchymal Stem Cells in Fibrotic Disease
Elie El Agha, Rafael Kramann, Rebekka K. Schneider, et al.
Cell stem cell (2017) Vol. 21, Iss. 2, pp. 166-177
Open Access | Times Cited: 378

Myofibroblasts
Boris Hinz
Experimental Eye Research (2015) Vol. 142, pp. 56-70
Closed Access | Times Cited: 368

Developmental signalling pathways in renal fibrosis: the roles of Notch, Wnt and Hedgehog
Maria Edeling, Grace Ragi, Shizheng Huang, et al.
Nature Reviews Nephrology (2016) Vol. 12, Iss. 7, pp. 426-439
Open Access | Times Cited: 359

The big five in fibrosis: Macrophages, myofibroblasts, matrix, mechanics, and miscommunication
Pardis Pakshir, Boris Hinz
Matrix Biology (2018) Vol. 68-69, pp. 81-93
Closed Access | Times Cited: 338

TGF-β-Induced Endothelial-Mesenchymal Transition in Fibrotic Diseases
Evangelia Pardali, Gonzalo Sánchez‐Duffhues, Maria Catalina Gomez‐Puerto, et al.
International Journal of Molecular Sciences (2017) Vol. 18, Iss. 10, pp. 2157-2157
Open Access | Times Cited: 309

Sickle cell disease: renal manifestations and mechanisms
Karl A. Nath, Robert P. Hebbel
Nature Reviews Nephrology (2015) Vol. 11, Iss. 3, pp. 161-171
Open Access | Times Cited: 301

Resolution of organ fibrosis
Joon-Il Jun, Lester F. Lau
Journal of Clinical Investigation (2018) Vol. 128, Iss. 1, pp. 97-107
Open Access | Times Cited: 300

Extracellular Matrix in Kidney Fibrosis: More Than Just a Scaffold
Roman D. Bülow, Peter Boor
Journal of Histochemistry & Cytochemistry (2019) Vol. 67, Iss. 9, pp. 643-661
Open Access | Times Cited: 297

Central role of dysregulation of TGF-β/Smad in CKD progression and potential targets of its treatment
Lin Chen, Tian Yang, Dewen Lu, et al.
Biomedicine & Pharmacotherapy (2018) Vol. 101, pp. 670-681
Open Access | Times Cited: 296

The role of myofibroblasts in wound healing
Boris Hinz
Current Research in Translational Medicine (2016) Vol. 64, Iss. 4, pp. 171-177
Closed Access | Times Cited: 286

Inflammation and renal fibrosis: Recent developments on key signaling molecules as potential therapeutic targets
Wenshan Lv, George W. Booz, Yangang Wang, et al.
European Journal of Pharmacology (2017) Vol. 820, pp. 65-76
Open Access | Times Cited: 273

What is damaging the kidney in lupus nephritis?
Anne Davidson
Nature Reviews Rheumatology (2015) Vol. 12, Iss. 3, pp. 143-153
Open Access | Times Cited: 269

Mechanisms of hypoxia signalling: new implications for nephrology
Johannes Schödel, Peter J. Ratcliffe
Nature Reviews Nephrology (2019) Vol. 15, Iss. 10, pp. 641-659
Closed Access | Times Cited: 268

The myofibroblast in wound healing and fibrosis: answered and unanswered questions
Marie‐Luce Bochaton‐Piallat, Giulio Gabbiani, Boris Hinz
F1000Research (2016) Vol. 5, pp. 752-752
Open Access | Times Cited: 262

Diverse origins of the myofibroblast—implications for kidney fibrosis
Lucas L. Falke, Shima Gholizadeh, Roel Goldschmeding, et al.
Nature Reviews Nephrology (2015) Vol. 11, Iss. 4, pp. 233-244
Closed Access | Times Cited: 257

Endothelial to Mesenchymal Transition (EndoMT) in the Pathogenesis of Human Fibrotic Diseases
Sonsoles Piera‐Velazquez, Fabian A. Mendoza, Sergio A. Jiménez
Journal of Clinical Medicine (2016) Vol. 5, Iss. 4, pp. 45-45
Open Access | Times Cited: 252

The next generation of therapeutics for chronic kidney disease
Matthew D. Breyer, Katalin Suszták
Nature Reviews Drug Discovery (2016) Vol. 15, Iss. 8, pp. 568-588
Open Access | Times Cited: 252

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