OpenAlex Citation Counts

OpenAlex Citations Logo

OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Brain distribution of dipeptide repeat proteins in frontotemporal lobar degeneration and motor neurone disease associated with expansions in C9ORF72
Yvonne S. Davidson, Holly Barker, Andrew Robinson, et al.
Acta Neuropathologica Communications (2014) Vol. 2, Iss. 1
Open Access | Times Cited: 111

Showing 1-25 of 111 citing articles:

Decoding ALS: from genes to mechanism
J. Paul Taylor, Robert H. Brown, Don W. Cleveland
Nature (2016) Vol. 539, Iss. 7628, pp. 197-206
Open Access | Times Cited: 1821

C9orf72-mediated ALS and FTD: multiple pathways to disease
Rubika Balendra, Adrian M. Isaacs
Nature Reviews Neurology (2018) Vol. 14, Iss. 9, pp. 544-558
Open Access | Times Cited: 616

Molecular neuropathology of frontotemporal dementia: insights into disease mechanisms from postmortem studies
Ian R. Mackenzie, Manuela Neumann
Journal of Neurochemistry (2016) Vol. 138, Iss. S1, pp. 54-70
Open Access | Times Cited: 315

TDP-43 Proteinopathy and ALS: Insights into Disease Mechanisms and Therapeutic Targets
Emma L. Scotter, Han-Jou Chen, Christopher E. Shaw
Neurotherapeutics (2015) Vol. 12, Iss. 2, pp. 352-363
Open Access | Times Cited: 308

C9orf72 BAC Transgenic Mice Display Typical Pathologic Features of ALS/FTD
Jacqueline G. O’Rourke, Laurent Bogdanik, A.K.M. Ghulam Muhammad, et al.
Neuron (2015) Vol. 88, Iss. 5, pp. 892-901
Open Access | Times Cited: 284

Molecular mechanisms underlying nucleotide repeat expansion disorders
Indranil Malik, Chase P. Kelley, Eric T. Wang, et al.
Nature Reviews Molecular Cell Biology (2021) Vol. 22, Iss. 9, pp. 589-607
Open Access | Times Cited: 263

The Role of Dipeptide Repeats in C9ORF72-Related ALS-FTD
Brian D. Freibaum, J. Paul Taylor
Frontiers in Molecular Neuroscience (2017) Vol. 10
Open Access | Times Cited: 244

C9orf72 expansions in frontotemporal dementia and amyotrophic lateral sclerosis
Jonathan D. Rohrer, Adrian M. Isaacs, Sarah Mizielinska, et al.
The Lancet Neurology (2015) Vol. 14, Iss. 3, pp. 291-301
Closed Access | Times Cited: 224

CRISPR–Cas9 screens in human cells and primary neurons identify modifiers of C9ORF72 dipeptide-repeat-protein toxicity
Nicholas J. Kramer, Michael S. Haney, David W. Morgens, et al.
Nature Genetics (2018) Vol. 50, Iss. 4, pp. 603-612
Open Access | Times Cited: 222

Quantitative analysis and clinico-pathological correlations of different dipeptide repeat protein pathologies in C9ORF72 mutation carriers
Ian R. Mackenzie, Petra Frick, Friedrich A. Grässer, et al.
Acta Neuropathologica (2015) Vol. 130, Iss. 6, pp. 845-861
Closed Access | Times Cited: 219

C9ORF72-ALS/FTD-associated poly(GR) binds Atp5a1 and compromises mitochondrial function in vivo
So Yoen Choi, Rodrigo López‐González, Gopinath Krishnan, et al.
Nature Neuroscience (2019) Vol. 22, Iss. 6, pp. 851-862
Open Access | Times Cited: 202

Review: An update on clinical, genetic and pathological aspects of frontotemporal lobar degenerations
Tammaryn Lashley, Jonathan D. Rohrer, Simon Mead, et al.
Neuropathology and Applied Neurobiology (2015) Vol. 41, Iss. 7, pp. 858-881
Closed Access | Times Cited: 190

Regulation of mRNA Translation in Neurons—A Matter of Life and Death
Mridu Kapur, Caitlin E. Monaghan, Susan L. Ackerman
Neuron (2017) Vol. 96, Iss. 3, pp. 616-637
Open Access | Times Cited: 190

RNA toxicity in non‐coding repeat expansion disorders
Bart Swinnen, Wim Robberecht, Ludo Van Den Bosch
The EMBO Journal (2019) Vol. 39, Iss. 1
Open Access | Times Cited: 147

p53 is a central regulator driving neurodegeneration caused by C9orf72 poly(PR)
Maya Maor-Nof, Zohar Shipony, Rodrigo López‐González, et al.
Cell (2021) Vol. 184, Iss. 3, pp. 689-708.e20
Open Access | Times Cited: 141

Distribution of dipeptide repeat proteins in cellular models and C9orf72 mutation cases suggests link to transcriptional silencing
Martin H. Schludi, Stephanie May, Friedrich A. Grässer, et al.
Acta Neuropathologica (2015) Vol. 130, Iss. 4, pp. 537-555
Open Access | Times Cited: 175

C9orf72 expansion disrupts ATM-mediated chromosomal break repair
Callum Walker, Saúl Herranz-Martín, Evangelia Karyka, et al.
Nature Neuroscience (2017) Vol. 20, Iss. 9, pp. 1225-1235
Open Access | Times Cited: 162

Antisense RNA foci in the motor neurons of C9ORF72-ALS patients are associated with TDP-43 proteinopathy
Johnathan Cooper‐Knock, Adrian Higginbottom, Matthew J. Stopford, et al.
Acta Neuropathologica (2015) Vol. 130, Iss. 1, pp. 63-75
Open Access | Times Cited: 159

There has been an awakening: Emerging mechanisms of C9orf72 mutations in FTD/ALS
Aaron D. Gitler, Hitomi Tsuiji
Brain Research (2016) Vol. 1647, pp. 19-29
Open Access | Times Cited: 148

Frontotemporal lobar degeneration: Pathogenesis, pathology and pathways to phenotype
David Mann, Julie S. Snowden
Brain Pathology (2017) Vol. 27, Iss. 6, pp. 723-736
Open Access | Times Cited: 137

The C9orf72 repeat expansion itself is methylated in ALS and FTLD patients
Zhengrui Xi, Ming Zhang, Amalia C. Bruni, et al.
Acta Neuropathologica (2015) Vol. 129, Iss. 5, pp. 715-727
Open Access | Times Cited: 136

Review: Neuropathology of non‐tau frontotemporal lobar degeneration
Manuela Neumann, Ian R. Mackenzie
Neuropathology and Applied Neurobiology (2018) Vol. 45, Iss. 1, pp. 19-40
Closed Access | Times Cited: 123

Early Cognitive, Structural, and Microstructural Changes in Presymptomatic C9orf72 Carriers Younger Than 40 Years
Anne Bertrand, Junhao Wen, Daisy Rinaldi, et al.
JAMA Neurology (2018) Vol. 75, Iss. 2, pp. 236-236
Open Access | Times Cited: 121

Novel clinical associations with specific C9ORF72 transcripts in patients with repeat expansions in C9ORF72
Marka van Blitterswijk, Tania F. Gendron, Matthew Baker, et al.
Acta Neuropathologica (2015) Vol. 130, Iss. 6, pp. 863-876
Open Access | Times Cited: 114

ALS biomarkers for therapy development: State of the field and future directions
Michael Benatar, Khrista Boylan, Andreas Jeromin, et al.
Muscle & Nerve (2015) Vol. 53, Iss. 2, pp. 169-182
Open Access | Times Cited: 109

Page 1 - Next Page

Scroll to top