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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Co-expression of C9orf72 related dipeptide-repeats over 1000 repeat units reveals age- and combination-specific phenotypic profiles in Drosophila
Ryan J. H. West, Joanne L. Sharpe, André Voelzmann, et al.
Acta Neuropathologica Communications (2020) Vol. 8, Iss. 1
Open Access | Times Cited: 33

Showing 1-25 of 33 citing articles:

CRISPR/Cas9-mediated excision of ALS/FTD-causing hexanucleotide repeat expansion in C9ORF72 rescues major disease mechanisms in vivo and in vitro
Katharina E. Meijboom, Abbas Abdallah, Nicholas P. Fordham, et al.
Nature Communications (2022) Vol. 13, Iss. 1
Open Access | Times Cited: 41
Physical activity as an exogenous risk factor for amyotrophic lateral sclerosis: a review of the evidence
Laura Chapman, Johnathan Cooper‐Knock, Pamela J. Shaw
Brain (2023) Vol. 146, Iss. 5, pp. 1745-1757
Open Access | Times Cited: 35
A bacterial artificial chromosome mouse model of amyotrophic lateral sclerosis manifests ‘space cadet syndrome’ on two FVB backgrounds
Shirlene Badger, Ian Coldicott, Ergita Kyrgiou-Balli, et al.
Disease Models & Mechanisms (2025) Vol. 18, Iss. 2
Open Access | Times Cited: 1
C9orf72 dipeptides disrupt the nucleocytoplasmic transport machinery and cause TDP-43 mislocalisation to the cytoplasm
Sarah Ryan, Sara Rollinson, Eleanor Hobbs, et al.
Scientific Reports (2022) Vol. 12, Iss. 1
Open Access | Times Cited: 37
Approaches to Gene Modulation Therapy for ALS
Katharina E. Meijboom, Robert H. Brown
Neurotherapeutics (2022) Vol. 19, Iss. 4, pp. 1159-1179
Open Access | Times Cited: 24
Intercellular transmission of pathogenic proteins in ALS: Exploring the pathogenic wave
Frederick J. Arnold, Ashlie Nguyen, Richard Bedlack, et al.
Neurobiology of Disease (2023) Vol. 184, pp. 106218-106218
Open Access | Times Cited: 16
Fly for ALS: Drosophila modeling on the route to amyotrophic lateral sclerosis modifiers
Francesco Liguori, Susanna Amadio, Cinzia Volonté
Cellular and Molecular Life Sciences (2021) Vol. 78, Iss. 17-18, pp. 6143-6160
Open Access | Times Cited: 30
Activation of the Keap1/Nrf2 pathway suppresses mitochondrial dysfunction, oxidative stress, and motor phenotypes inC9orf72ALS/FTD models
Wing Hei Au, Leonor Miller‐Fleming, Álvaro Sánchez-Martínez, et al.
Life Science Alliance (2024) Vol. 7, Iss. 9, pp. e202402853-e202402853
Open Access | Times Cited: 4
Altered Phase Separation and Cellular Impact in C9orf72-Linked ALS/FTD
Daniel A. Solomon, Rebekah Smikle, Matthew J. Reid, et al.
Frontiers in Cellular Neuroscience (2021) Vol. 15
Open Access | Times Cited: 26
Multiple pathways of toxicity induced by C9orf72 dipeptide repeat aggregates and G4C2 RNA in a cellular model
Frédéric Frottin, Manuela Pérez‐Berlanga, F. Ulrich Hartl, et al.
eLife (2021) Vol. 10
Open Access | Times Cited: 25
Drosophila Primary Neuronal Cultures as a Useful Cellular Model to Study and Image Axonal Transport
André Voelzmann, Natalia Sánchez‐Soriano
Methods in molecular biology (2022), pp. 429-449
Open Access | Times Cited: 13
Artificial microRNA suppresses C9ORF72 variants and decreases toxic dipeptide repeat proteins in vivo
Gabriela Toro Cabrera, Katharina E. Meijboom, Abbas Abdallah, et al.
Gene Therapy (2023) Vol. 31, Iss. 3-4, pp. 105-118
Open Access | Times Cited: 7
Opto-controlled C9orf72 poly-PR forms anisotropic condensates causative of TDP-43 pathology in the nucleus
Rachel E. Hodgson, Jessica A Rayment, Wan-Ping Huang, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2024)
Open Access | Times Cited: 2
Molecular mechanisms and therapeutic strategies for neuromuscular diseases
Alberto A. Zambon, Falzone Yuri Matteo, Bolino Alessandra, et al.
Cellular and Molecular Life Sciences (2024) Vol. 81, Iss. 1
Open Access | Times Cited: 2
Promising animal models for amyotrophic lateral sclerosis drug discovery: a comprehensive update
Léa Lescouzères, Scott B. Patten
Expert Opinion on Drug Discovery (2024) Vol. 19, Iss. 10, pp. 1213-1233
Open Access | Times Cited: 2
C9orf72 dipeptides activate the NLRP3 inflammasome
Jack Rivers‐Auty, Christopher Hoyle, Ayesha Pointer, et al.
Brain Communications (2024) Vol. 6, Iss. 5
Open Access | Times Cited: 2
Exploring the alternative: Fish, flies and worms as preclinical models for ALS
Elke Braems, Paraskevi Tziortzouda, Ludo Van Den Bosch
Neuroscience Letters (2021) Vol. 759, pp. 136041-136041
Open Access | Times Cited: 13
Senataxin helicase, the causal gene defect in ALS4, is a significant modifier of C9orf72 ALS G4C2 and arginine-containing dipeptide repeat toxicity
Craig L. Bennett, Somasish Ghosh Dastidar, Frederick J. Arnold, et al.
Acta Neuropathologica Communications (2023) Vol. 11, Iss. 1
Open Access | Times Cited: 5
Toxicity ofC9orf72-associated dipeptide repeat peptides is modified by commonly used protein tags
Javier Morón-Oset, Lilly KS Fischer, Mireia Carcolé, et al.
Life Science Alliance (2023) Vol. 6, Iss. 9, pp. e202201739-e202201739
Open Access | Times Cited: 4
Advances in the Structure of GGGGCC Repeat RNA Sequence and Its Interaction with Small Molecules and Protein Partners
Xiaole Liu, Xinyue Zhao, Jinhan He, et al.
Molecules (2023) Vol. 28, Iss. 15, pp. 5801-5801
Open Access | Times Cited: 4
Repeat length of C9orf72-associated glycine–alanine polypeptides affects their toxicity
Javier Morón-Oset, Lilly Katharina Sophie Fischer, Nathalie Jauré, et al.
Acta Neuropathologica Communications (2023) Vol. 11, Iss. 1
Open Access | Times Cited: 4
C9orf72 proline-arginine dipeptide repeats disrupt the proteasome and perturb proteolytic activities
Yifan Zhang, S. Nelson, Ashley P Viera Ortiz, et al.
Journal of Neuropathology & Experimental Neurology (2023) Vol. 82, Iss. 11, pp. 901-910
Open Access | Times Cited: 4
C9orf72 poly-PR forms anisotropic condensates causative of nuclear TDP-43 pathology
Rachel E. Hodgson, Jessica A Rayment, Wan-Ping Huang, et al.
iScience (2024) Vol. 27, Iss. 10, pp. 110937-110937
Open Access | Times Cited: 1
Modeling C9orf72-Related Frontotemporal Dementia and Amyotrophic Lateral Sclerosis in Drosophila
Joanne L. Sharpe, Nikki S. Harper, Duncan R. Garner, et al.
Frontiers in Cellular Neuroscience (2021) Vol. 15
Open Access | Times Cited: 7
Phenotype and management of neurologic intronic repeat disorders (NIRDs)
Josef Finsterer
Revue Neurologique (2022) Vol. 179, Iss. 3, pp. 173-182
Closed Access | Times Cited: 4
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