OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Overexpression screen of chromosome 21 genes reveals modulators of Sonic hedgehog signaling relevant to Down syndrome
Anna J. Moyer, Fabian‐Xosé Fernandez, Yicong Li, et al.
Disease Models & Mechanisms (2023) Vol. 16, Iss. 4
Open Access | Times Cited: 9

Showing 9 citing articles:

Mouse Models of Down Syndrome
Andrew Folz, Kourtney Sloan, Randall J. Roper
(2025), pp. 1-43
Closed Access

Simple Worm, Complex Disorder: Studying Down Syndrome Mechanisms in the Unassuming Model C. elegans
Sergio Sánchez, Jonathan T. Pierce
(2025), pp. 87-149
Closed Access

Patterns of Aneuploidy and Signaling Consequences in Cancer
Nadja Zhakula-Kostadinova, Alison M. Taylor
Cancer Research (2024) Vol. 84, Iss. 16, pp. 2575-2587
Open Access | Times Cited: 3

Integration of ATAC-seq and RNA-seq identifies MX1-mediated AP-1 transcriptional regulation as a therapeutic target for Down syndrome
Zhenglong Guo, Yongchang Zhu, Hai Xiao, et al.
Biological Research (2023) Vol. 56, Iss. 1
Open Access | Times Cited: 5

Network Pharmacology Identifies Intersection Genes of Apigenin and Naringenin in Down Syndrome as Potential Therapeutic Targets
Mohd Amir, Shabana Shafi, Shahida Parveen, et al.
Pharmaceuticals (2024) Vol. 17, Iss. 8, pp. 1090-1090
Open Access | Times Cited: 1

Comparative analysis of craniofacial shape in two mouse models of Down syndrome: Ts65Dn and TcMAC21
Nandini Singh, Joan T. Richtsmeier, Roger H. Reeves
Journal of Anatomy (2024) Vol. 244, Iss. 6, pp. 1007-1014
Closed Access

Increased endothelial sclerostin caused by elevated DSCAM mediates multiple trisomy 21 phenotypes
David McKean, Qi Zhang, Priyanka Narayan, et al.
Journal of Clinical Investigation (2024) Vol. 134, Iss. 11
Open Access

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