OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Transcriptome Analysis Reveals That Ascorbic Acid Treatment Enhances the Cold Tolerance of Tea Plants through Cell Wall Remodeling
Qianyuan Fu, Hongli Cao, Lu Wang, et al.
International Journal of Molecular Sciences (2023) Vol. 24, Iss. 12, pp. 10059-10059
Open Access | Times Cited: 11

Showing 11 citing articles:

Inhibition of wound healing in fresh-cut potatoes by ascorbic acid is associated with control of the levels of reactive oxygen species and the AsA-GSH cycle
Fuhui Zhou, Dongying Xu, Siguo Xiong, et al.
Scientia Horticulturae (2023) Vol. 323, pp. 112472-112472
Closed Access | Times Cited: 23

Transcriptomics and Plant Hormone Analysis Reveal the Mechanism of Branching Angle Formation in Tea Plants (Camellia sinensis)
Jinping Zhu, Xiaoman Li, Jianyan Huang, et al.
International Journal of Molecular Sciences (2025) Vol. 26, Iss. 2, pp. 604-604
Open Access | Times Cited: 1

Analysis of the two-component system gene family and the positive role of CsRR5 in cold stress response in tea plants
Xiaobin Dong, Changqing Ding, Xuening Zhang, et al.
Plant Physiology and Biochemistry (2025) Vol. 222, pp. 109739-109739
Closed Access

In vitro VS in vivo transcriptional approach provides new insight of temporal cold response which works without root system in tea plant (Camellia sinensis (L.) Kuntze)
Kedong Da, Samarina LS, Malyukova LS, et al.
Plant Physiology and Biochemistry (2025), pp. 109934-109934
Closed Access

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