OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Cytogenetic characterisation and proteomic profiling of the Imatinib-resistant cell line KCL22-R
Julia Rosenhahn, Anja Weise, Susanne Michel, et al.
International Journal of Oncology (2007)
Open Access | Times Cited: 15

Showing 15 citing articles:

Survival outcomes for clonal evolution in chronic myeloid leukemia patients on second generation tyrosine kinase inhibitor therapy
Dushyant Verma, Hagop M. Kantarjian, Jianqin Shan, et al.
Cancer (2010) Vol. 116, Iss. 11, pp. 2673-2681
Open Access | Times Cited: 65

BCR-ABL Gene Expression Is Required for Its Mutations in a Novel KCL-22 Cell Culture Model for Acquired Resistance of Chronic Myelogenous Leukemia
Hongfeng Yuan, Zhiqiang Wang, Chunggang Gao, et al.
Journal of Biological Chemistry (2009) Vol. 285, Iss. 7, pp. 5085-5096
Open Access | Times Cited: 47

Evolutionary karyotypic theory of cancer versus conventional cancer gene mutation theory
Aleksei A. Stepanenko, V. M. Kavsan
Biopolymers and Cell (2012) Vol. 28, Iss. 4, pp. 267-280
Open Access | Times Cited: 26

Clonal Evolution and Blast Crisis Correlate with Enhanced Proteolytic Activity of Separase in BCR-ABL b3a2 Fusion Type CML under Imatinib Therapy
Wiltrud Haaß, Helga Kleiner, Christel Weiß, et al.
PLoS ONE (2015) Vol. 10, Iss. 6, pp. e0129648-e0129648
Open Access | Times Cited: 18

Sensitivity of imatinib-resistant T315I BCR-ABL CML to a synergistic combination of ponatinib and forskolin treatment
Derrick M. Oaxaca, Sun Ah Yang-Reid, Jeremy A. Ross, et al.
Tumor Biology (2016) Vol. 37, Iss. 9, pp. 12643-12654
Open Access | Times Cited: 15

Gaining insights into the Bcr-Abl activity-independent mechanisms of resistance to imatinib mesylate in KCL22 cells: A comparative proteomic approach
Irene Colavita, Nicola Esposito, Rosanna Martinelli, et al.
Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics (2010) Vol. 1804, Iss. 10, pp. 1974-1987
Closed Access | Times Cited: 18

The Proteolytic Activity of Separase in BCR-ABL-Positive Cells Is Increased by Imatinib
Wiltrud Haaß, Michael Stehle, Stefanie Nittka, et al.
PLoS ONE (2012) Vol. 7, Iss. 8, pp. e42863-e42863
Open Access | Times Cited: 17

Molecular Cytogenetic Characterization of the Human Cerebral Microvessel Endothelial Cell Line hCMEC/D3
Hasmik Mkrtchyan, Stefan Scheler, Isaac A. Klein, et al.
Cytogenetic and Genome Research (2009) Vol. 126, Iss. 4, pp. 313-317
Closed Access | Times Cited: 13

Genetic counseling in carriers of reciprocal translocations involving two autosomes
Hamid Pour-Jafari, Safieh Ghahramani, Bahareh Pourjafari, et al.
Indian journal of human genetics (2012) Vol. 18, Iss. 2, pp. 250-250
Open Access | Times Cited: 10

RUNX1T1 is overexpressed in imatinib mesylate-resistant cells
Binato
Molecular Medicine Reports (2009) Vol. 2, Iss. 4
Open Access | Times Cited: 7

Proteomic approaches for investigation of therapy resistance in cancer
Hermann Lage
PROTEOMICS - CLINICAL APPLICATIONS (2009) Vol. 3, Iss. 8, pp. 883-911
Closed Access | Times Cited: 6

Insights into apoptotic proteins in chemotherapy: quantification techniques and informing therapy choice
Janine Naß, Thomas Efferth
Expert Review of Proteomics (2018) Vol. 15, Iss. 5, pp. 413-429
Closed Access | Times Cited: 3

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