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OpenAlex Citation Counts

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OpenAlex is a bibliographic catalogue of scientific papers, authors and institutions accessible in open access mode, named after the Library of Alexandria. It's citation coverage is excellent and I hope you will find utility in this listing of citing articles!

If you click the article title, you'll navigate to the article, as listed in CrossRef. If you click the Open Access links, you'll navigate to the "best Open Access location". Clicking the citation count will open this listing for that article. Lastly at the bottom of the page, you'll find basic pagination options.

Requested Article:

Cross-species analysis of LZTR1 loss-of-function mutants demonstrates dependency to RIT1 orthologs
Antonio Cuevas-Navarro, Laura Rodríguez-Muñoz, Joaquím Grego‐Bessa, et al.
eLife (2022) Vol. 11
Open Access | Times Cited: 16

Showing 16 citing articles:

Risk of meningomyelocele mediated by the common 22q11.2 deletion
Keng Ioi Vong, Sangmoon Lee, Kit Sing Au, et al.
Science (2024) Vol. 384, Iss. 6695, pp. 584-590
Closed Access | Times Cited: 6
Impaired Proteolysis of Noncanonical RAS Proteins Drives Clonal Hematopoietic Transformation
Sisi Chen, Rahul S. Vedula, Antonio Cuevas-Navarro, et al.
Cancer Discovery (2022) Vol. 12, Iss. 10, pp. 2434-2453
Open Access | Times Cited: 23
RAS-dependent RAF-MAPK hyperactivation by pathogenic RIT1 is a therapeutic target in Noonan syndrome–associated cardiac hypertrophy
Antonio Cuevas-Navarro, Morgan E. Wagner, Richard Van, et al.
Science Advances (2023) Vol. 9, Iss. 28
Open Access | Times Cited: 13
Preclinical evaluation of CRISPR-based therapies for Noonan syndrome caused by deep-intronic LZTR1 variants
Carolin Knauer, Henrike Haltern, Eric Schoger, et al.
Molecular Therapy — Nucleic Acids (2024) Vol. 35, Iss. 1, pp. 102123-102123
Open Access | Times Cited: 5
Mutation-induced LZTR1 polymerization provokes cardiac pathology in recessive Noonan syndrome
Alexandra Viktoria Busley, Óscar Gutiérrez‐Gutiérrez, Elke Hammer, et al.
Cell Reports (2024) Vol. 43, Iss. 7, pp. 114448-114448
Open Access | Times Cited: 4
The tumor suppressor LZTR1: Its expression, purification and characterization
Yifang Sun, Yuxuan Jiang, Meng Zhang, et al.
Protein Expression and Purification (2025), pp. 106716-106716
Closed Access
Defective protein degradation in genetic disorders
Pau Castel
Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease (2022) Vol. 1868, Iss. 5, pp. 166366-166366
Open Access | Times Cited: 5
LZTR1 polymerization provokes cardiac pathology in recessive Noonan syndrome
Alexandra Viktoria Busley, Óscar Gutiérrez‐Gutiérrez, Elke Hammer, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2023)
Open Access | Times Cited: 2
Non-Mammalian Models for Understanding Neurological Defects in RASopathies
Mario Rodríguez-Martín, Juan Báez-Flores, Vanessa Ribes, et al.
Biomedicines (2024) Vol. 12, Iss. 4, pp. 841-841
Open Access
The deubiquitinase USP9X regulates RIT1 protein abundance and oncogenic phenotypes
Amanda K. Riley, Michael Grant, Aidan Snell, et al.
iScience (2024) Vol. 27, Iss. 8, pp. 110499-110499
Open Access
Typical NF2 and LTZR1 Mutations Are Retained in an Immortalized Human Schwann Cell Model of Schwannomatosis
Valentina Melfi, Tasnim Mohamed, Alessandra Colciago, et al.
Heliyon (2024) Vol. 10, Iss. 19, pp. e38957-e38957
Open Access
The Noncanonical RAS/MAPK Pathway and the RASopathies
Stephanie Mo, Pau Castel
(2024), pp. 427-456
Closed Access
The deubiquitinase USP9X regulates RIT1 protein abundance and oncogenic phenotypes
Amanda K. Riley, Michael Grant, Aidan Snell, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2023)
Open Access
Biallelic LZTR1 variants in a 49‐year‐old woman with hypertrophic cardiomyopathy: A clue for considering LZTR1 in adults
Giuseppe Di Stolfo, Antonio Petracca, Ester Maria Lucia Bevere, et al.
American Journal of Medical Genetics Part A (2023) Vol. 194, Iss. 5
Open Access
Exploring the Regulation of Cdc42 Stability and Turnover in Yeast
Beatriz González, Martí Aldea, Paul J. Cullen
bioRxiv (Cold Spring Harbor Laboratory) (2022)
Open Access
Ras-dependent RAF-MAPK hyperactivation by pathogenic RIT1 is a therapeutic target in Noonan syndrome-associated cardiac hypertrophy
Antonio Cuevas-Navarro, Morgan E. Wagner, Richard Van, et al.
bioRxiv (Cold Spring Harbor Laboratory) (2022)
Open Access
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